Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Jason J. Fritz"'
Autor:
Ranjita Betarbet, Leah R. Anderson, Marla Gearing, Tiffany R. Hodges, Jason J. Fritz, James J. Lah, Allan I. Levey
Publikováno v:
Neurobiology of Disease, Vol 31, Iss 3, Pp 309-315 (2008)
Fas-associated factor 1 or FAF1 is a Fas-binding protein implicated in apoptosis. FAF1 is the product of a gene at PARK 10 locus on chromosome 1p32, a locus associated with late-onset PD [Hicks, A.A., Petursson, H., Jonsson, T., Stefansson, H., Johan
Externí odkaz:
https://doaj.org/article/7c9fcdd98d094fa0a18df4435acfc15c
Autor:
Amarallys F. Cintron, Jason J. Fritz, James J. Lah, Lary C. Walker, Tsuyoshi Hamaguchi, Jeromy Dooyema, Harry LeVine, Rebecca F. Rosen, Mathias Jucker
Publikováno v:
Journal of Neurochemistry. 120:660-666
J. Neurochem. (2012) 120, 660–666. Abstract Deposition of the amyloid-β (Aβ) peptide in senile plaques and cerebral Aβ angiopathy (CAA) can be stimulated in Aβ-precursor protein (APP)-transgenic mice by the intracerebral injection of dilute bra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3ac84b10372f4e6c7837edcb51120cf9
https://doi.org/10.1111/j.1471-4159.2011.07551.x
https://doi.org/10.1111/j.1471-4159.2011.07551.x
Publikováno v:
The Journal of Neuroscience. 30:4190-4196
Alzheimer's disease (AD) is a progressive neurological disorder that causes dementia and poses a major public health crisis as the population ages. Aberrant processing of the amyloid precursor protein (APP) is strongly implicated as a proximal event
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e219428bd19f28f5fb6c0c1b9973bfdf
https://doi.org/10.1523/jneurosci.6393-09.2010
https://doi.org/10.1523/jneurosci.6393-09.2010
Autor:
Tiffany R. Hodges, Marla Gearing, Leah R. Anderson, Ranjita Betarbet, James J. Lah, Jason J. Fritz, Allan I. Levey
Publikováno v:
Neurobiology of Disease, Vol 31, Iss 3, Pp 309-315 (2008)
Fas-associated factor 1 or FAF1 is a Fas-binding protein implicated in apoptosis. FAF1 is the product of a gene at PARK 10 locus on chromosome 1p32, a locus associated with late-onset PD [Hicks, A.A., Petursson, H., Jonsson, T., Stefansson, H., Johan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a69260602a1b2094829ae80847930ef4
http://www.sciencedirect.com/science/article/pii/S0969996108000983
http://www.sciencedirect.com/science/article/pii/S0969996108000983
Autor:
Sylvia E. Perez, Stephen W. Scheff, Scott E. Counts, Muhammad Nadeem, Elliott J. Mufson, Stephen D. Ginsberg, Joanne Wuu, Bin He, James J. Lah, Sue Leurgans, Jason J. Fritz
Hippocampal precursor of nerve growth factor (proNGF)/NGF signaling occurs in conjunction with β-amyloid (Aβ) accumulations in Alzheimer disease (AD). To assess the involvement of this pathway in AD progression, we quantified these proteins and the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2558ed614a8c0da3f2708eeaf743da1
https://europepmc.org/articles/PMC3481187/
https://europepmc.org/articles/PMC3481187/
Autor:
Nirjari V. Dalal, Adam L. Orr, Jason J. Fritz, Ranjita Betarbet, Craig J. Heilman, Jeremy H. Herskowitz, Allan I. Levey, Leah A Roesch, Elaine L. Pranski, James J. Lah
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 9, Iss 1, p 67 (2012)
Journal of Neuroinflammation, Vol 9, Iss 1, p 67 (2012)
Background The RING domain-containing protein RING finger protein 11 (RNF11) is a member of the A20 ubiquitin-editing protein complex and modulates peripheral NF-κB signaling. RNF11 is robustly expressed in neurons and colocalizes with a population
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59fbcbcbc7c1a1d42c4be5d6abb5ac94
https://doi.org/10.1186/1742-2094-9-67
https://doi.org/10.1186/1742-2094-9-67
Autor:
Junmin Peng, Dongmei Cheng, Jason J. Fritz, Craig J. Heilman, Jonathan D. Glass, James J. Lah, Allan I. Levey, Duc M. Duong, Howard D. Rees, Qiangwei Xia, Joanne Wuu, Nicholas T. Seyfried, Yair M. Gozal, Deborah S. Cooper, Marla Gearing
Publikováno v:
Molecular Neurodegeneration, Vol 6, Iss 1, p 82 (2011)
Molecular Neurodegeneration
Molecular Neurodegeneration
Background Detergent-insoluble protein accumulation and aggregation in the brain is one of the pathological hallmarks of neurodegenerative diseases. Here, we describe the identification of septin 11 (SEPT11), an enriched component of detergent-resist
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b277ae1df646c39154da814f5af1c07b
https://doi.org/10.1186/1750-1326-6-82
https://doi.org/10.1186/1750-1326-6-82
Autor:
Kenneth A. Moore, Sylvia E. Perez, Jason J. Fritz, Orly Lazarov, James J. Lah, Yuan Shih Hu, Scott E. Counts, Brian M. Berg, Elliott J. Mufson, Bin He
Publikováno v:
Journal of Neuroscience Research.
Epidemiological and clinical trial findings suggest that consumption of docosahexaenoic acid (DHA) lowers the risk of Alzheimer's disease (AD). We examined the effects of short-term (3 months) DHA enriched diet on plaque deposition and synaptic defec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::160e47a708339678a47a0d1a9f998479
https://doi.org/10.1002/jnr.22266
https://doi.org/10.1002/jnr.22266
Autor:
Lary C. Walker, Jason J. Fritz, Anthony W.S. Chan, James J. Lah, Alexey Bogush, Rebecca F. Rosen, Stephanie Stephens, Michelle E. Ehrlich, Sam Gandy, Howard D. Rees, Stephanie C. Carter, Yol Smith, Allan I. Levey
Publikováno v:
Alzheimer's & Dementia. 2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::877a40350c1b4a5233acac9ead0f49c2
https://doi.org/10.1016/j.jalz.2006.05.392
https://doi.org/10.1016/j.jalz.2006.05.392
Autor:
Jason J. Fritz, Sara E. Dodson, Katrin Offe, James J. Lah, James T. Shoemaker, Allan I. Levey, Marla Gearing
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 26(5)
Alzheimer's disease (AD) is a neurodegenerative disorder characterized by progressive cognitive decline and neuropathological changes, including the deposition of amyloid beta (Abeta) in senile plaques. The mechanisms causing the disease and Abeta ac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f4bd86a7074277f5d7d9483d2301d2ed
https://pubmed.ncbi.nlm.nih.gov/16452683
https://pubmed.ncbi.nlm.nih.gov/16452683