Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Jasmin I Maier"'
Autor:
Mako Yasuda-Yamahara, Manuel Rogg, Kosuke Yamahara, Jasmin I Maier, Tobias B Huber, Christoph Schell
Publikováno v:
PLoS ONE, Vol 13, Iss 7, p e0200487 (2018)
Podocytes are highly-specialized epithelial cells essentially required for the generation and the maintenance of the kidney filtration barrier. This elementary function is directly based on an elaborated cytoskeletal apparatus establishing a complex
Externí odkaz:
https://doaj.org/article/417c490aeecf4518a44a609e89396886
Autor:
Manuel Rogg, Jasmin I. Maier, Martin Helmstädter, Alena Sammarco, Felix Kliewe, Oliver Kretz, Lisa Weißer, Clara Van Wymersch, Karla Findeisen, Anna L. Koessinger, Olga Tsoy, Jan Baumbach, Markus Grabbert, Martin Werner, Tobias B. Huber, Nicole Endlich, Oliver Schilling, Christoph Schell
Publikováno v:
Cells, Vol 12, Iss 13, p 1795 (2023)
Glomerular disease due to podocyte malfunction is a major factor in the pathogenesis of chronic kidney disease. Identification of podocyte-specific signaling pathways is therefore a prerequisite to characterizing relevant disease pathways and develop
Externí odkaz:
https://doaj.org/article/65be523bab45463eaad86d94233ee798
Autor:
Jasmin I. Maier, Manuel Rogg, Martin Helmstädter, Alena Sammarco, Oliver Schilling, Benedikt Sabass, Jeffrey H. Miner, Jörn Dengjel, Gerd Walz, Martin Werner, Tobias B. Huber, Christoph Schell
Publikováno v:
Cell Reports, Vol 34, Iss 12, Pp 108883- (2021)
Summary: The integrity of the kidney filtration barrier essentially relies on the balanced interplay of podocytes and the glomerular basement membrane (GBM). Here, we show by analysis of in vitro and in vivo models that a loss of the podocyte-specifi
Externí odkaz:
https://doaj.org/article/726d0e2f26e7481c9784ecf97f6f2b07
Autor:
Manuel Rogg, Jasmin I. Maier, Markus Ehle, Alena Sammarco, Oliver Schilling, Martin Werner, Christoph Schell
Publikováno v:
Cells, Vol 11, Iss 8, p 1259 (2022)
Steroid-resistant nephrotic syndrome (SRNS) frequently leads to end-stage renal disease, ultimately requiring kidney replacement therapies. SRNS is often caused by hereditary monogenic mutations, specifically affecting specialized epithelial cells (p
Externí odkaz:
https://doaj.org/article/7b60595e4f2e463ea102abc45d00481b
Autor:
Schell, Manuel Rogg, Jasmin I. Maier, Martin Helmstädter, Alena Sammarco, Felix Kliewe, Oliver Kretz, Lisa Weißer, Clara Van Wymersch, Karla Findeisen, Anna L. Koessinger, Olga Tsoy, Jan Baumbach, Markus Grabbert, Martin Werner, Tobias B. Huber, Nicole Endlich, Oliver Schilling, Christoph
Publikováno v:
Cells; Volume 12; Issue 13; Pages: 1795
Glomerular disease due to podocyte malfunction is a major factor in the pathogenesis of chronic kidney disease. Identification of podocyte-specific signaling pathways is therefore a prerequisite to characterizing relevant disease pathways and develop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=multidiscipl::a969df7eaccafba20d882d3836b14016
Autor:
Jasmin I. Maier, Manuel Rogg, Martin Helmstädter, Alena Sammarco, Gerd Walz, Martin Werner, Christoph Schell
Publikováno v:
Cells, Vol 10, Iss 6, p 1509 (2021)
Glomerular kidney disease causing nephrotic syndrome is a complex systemic disorder and is associated with significant morbidity in affected patient populations. Despite its clinical relevance, well-established models are largely missing to further e
Externí odkaz:
https://doaj.org/article/fe3ddd397b274e8890f60606c2e6d7e3
Autor:
Manuel Rogg, Jasmin I. Maier, Clara Van Wymersch, Martin Helmstädter, Alena Sammarco, Maja Lindenmeyer, Paulina Zareba, Eloi Montanez, Gerd Walz, Martin Werner, Nicole Endlich, Thomas Benzing, Tobias B. Huber, Christoph Schell
Publikováno v:
J Am Soc Nephrol
BACKGROUND: The cell-matrix adhesion between podocytes and the glomerular basement membrane is essential for the integrity of the kidney’s filtration barrier. Despite increasing knowledge about the complexity of integrin adhesion complexes, an unde
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac28ef8d93627075668e3b5696733ac6
https://doi.org/10.1681/asn.2021101319
https://doi.org/10.1681/asn.2021101319
Autor:
Schell, Jasmin I. Maier, Manuel Rogg, Martin Helmstädter, Alena Sammarco, Gerd Walz, Martin Werner, Christoph
Publikováno v:
Cells; Volume 10; Issue 6; Pages: 1509
Glomerular kidney disease causing nephrotic syndrome is a complex systemic disorder and is associated with significant morbidity in affected patient populations. Despite its clinical relevance, well-established models are largely missing to further e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=multidiscipl::3432fdf8471ee4c350e5c6c8ee490f73
Autor:
Dominik Sellung, Oliver Kretz, Nicole Endlich, Tobias B. Huber, Ahmed Abed, Gerd Walz, Martin L. Biniossek, Karoline Reiche, Christoph Schell, Martin Helmstädter, Martin Werner, August Sigle, Jasmin I. Maier, Nadine Artelt, Oliver Schilling, Robert Dotzauer, Manuel Rogg, Mako Yasuda-Yamahara, Patrick Dinse, Alena Sammarco
Publikováno v:
American Society of Nephrology
J Am Soc Nephrol
J Am Soc Nephrol
BACKGROUND: Previous research demonstrated that small Rho GTPases, modulators of the actin cytoskeleton, are drivers of podocyte foot-process effacement in glomerular diseases, such as FSGS. However, a comprehensive understanding of the regulatory ne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::657803b6fe24bceb8a7f61109f7bf790
Autor:
Tobias B. Huber, Manuel Rogg, Mako Yasuda-Yamahara, Christoph Schell, Kosuke Yamahara, Jasmin I. Maier
Publikováno v:
PLOS ONE
PLoS ONE
PLoS ONE, Vol 13, Iss 7, p e0200487 (2018)
PLoS ONE
PLoS ONE, Vol 13, Iss 7, p e0200487 (2018)
Podocytes are highly-specialized epithelial cells essentially required for the generation and the maintenance of the kidney filtration barrier. This elementary function is directly based on an elaborated cytoskeletal apparatus establishing a complex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::037bd7b4eac0f82541593e69b3d0e0b0