Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Janice M. Nigro"'
Autor:
Barbara Klink, Hrvoje Miletic, Daniel Stieber, Peter C. Huszthy, Jaime Alberto Campos Valenzuela, Jörg Balss, Jian Wang, Manja Schubert, Per Øystein Sakariassen, Terje Sundstrøm, Anja Torsvik, Mads Aarhus, Rupavathana Mahesparan, Andreas von Deimling, Lars Kaderali, Simone P. Niclou, Evelin Schröck, Rolf Bjerkvig, Janice M. Nigro
Publikováno v:
PLoS ONE, Vol 8, Iss 8 (2013)
Externí odkaz:
https://doaj.org/article/49ff2cf189664a1a83fc2593441df7e8
Autor:
Kenneth D. Aldape, Burt G. Feuerstein, W.K. Alfred Yung, Dimpy Koul, Edward Pan, Mingang Chen, Natalie Ozburn, Chandi Griffin, Howard Colman, Ivan Smirnov, Li Zhang, Anjan Misra, Janice M. Nigro
5 Tables and 3 Figures from Integrated Array-Comparative Genomic Hybridization and Expression Array Profiles Identify Clinically Relevant Molecular Subtypes of Glioblastoma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::810bc5c8cb988d6aabd70a602c98a581
https://doi.org/10.1158/0008-5472.22364790
https://doi.org/10.1158/0008-5472.22364790
Autor:
Kenneth D. Aldape, Burt G. Feuerstein, W.K. Alfred Yung, Dimpy Koul, Edward Pan, Mingang Chen, Natalie Ozburn, Chandi Griffin, Howard Colman, Ivan Smirnov, Li Zhang, Anjan Misra, Janice M. Nigro
Glioblastoma, the most aggressive primary brain tumor in humans, exhibits a large degree of molecular heterogeneity. Understanding the molecular pathology of a tumor and its linkage to behavior is an important foundation for developing and evaluating
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2152e0238aaa5701158bbb572fabb090
https://doi.org/10.1158/0008-5472.c.6494364
https://doi.org/10.1158/0008-5472.c.6494364
Autor:
Khalil Abou-El-Ardat, Nadin D. Exner, Jaime Alberto Campos Valenzuela, Hrvoje Miletic, Peter C. Huszthy, Barbara Klink, Petra M. Radehaus, Janice M. Nigro, Lars Kaderali, Rolf Bjerkvig, Evelin Schröck
Publikováno v:
Oncotarget
We previously reported the establishment of a rare xenograft derived from a recurrent oligodendroglioma with 1p/19q codeletion. Here, we analyzed in detail the exome sequencing datasets from the recurrent oligodendroglioma (WHO grade III, recurrent O
Autor:
Barbara Klink, Hrvoje Miletic, Daniel Stieber, Peter C Huszthy, Jaime Alberto Campos Valenzuela, Jörg Balss, Jian Wang, Manja Schubert, Per Øystein Sakariassen, Terje Sundstrøm, Anja Torsvik, Mads Aarhus, Rupavathana Mahesparan, Andreas von Deimling, Lars Kaderali, Simone P Niclou, Evelin Schröck, Rolf Bjerkvig, Janice M Nigro
Publikováno v:
PLoS ONE, Vol 8, Iss 3, p e59773 (2013)
Oligodendroglioma poses a biological conundrum for malignant adult human gliomas: it is a tumor type that is universally incurable for patients, and yet, only a few of the human tumors have been established as cell populations in vitro or as intracra
Externí odkaz:
https://doaj.org/article/0fde4b9e5f4a4bf1977638549aa2787d
Autor:
Sophie Eisenreich, Khalil Abou-El-Ardat, Karol Szafranski, Jaime A Campos Valenzuela, Andreas Rump, Janice M Nigro, Rolf Bjerkvig, Eva-Maria Gerlach, Karl Hackmann, Evelin Schröck, Dietmar Krex, Lars Kaderali, Gabriele Schackert, Matthias Platzer, Barbara Klink
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e76623 (2013)
Oligodendroglial tumors form a distinct subgroup of gliomas, characterized by a better response to treatment and prolonged overall survival. Most oligodendrogliomas and also some oligoastrocytomas are characterized by a unique and typical unbalanced
Externí odkaz:
https://doaj.org/article/801c70ba21a348528b99e6c36c4f4533
Autor:
Krishna M Talasila, Narve Brekka, Kjersti Mangseth, Daniel Stieber, Lasse Evensen, Gro V Rosland, Anja Torsvik, Marek Wagner, Simone P Niclou, Rupavathana Mahesparan, Olav K Vintermyr, Rolf Bjerkvig, Janice M Nigro, Hrvoje Miletic
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e81183 (2013)
Two of the signature genetic events that occur in human gliomas, EGFR amplification and IDH mutation, are poorly represented in experimental models in vitro. EGFR amplification, for example, occurs in 40 to 50% of GBM, and yet, EGFR amplification is
Externí odkaz:
https://doaj.org/article/e11b2a7f9c3149ffb7dc882e61c265ab
Autor:
Narve Brekka, Olav Karsten Vintermyr, Per Øystein Sakariassen, Philipp Euskirchen, Anke Soentgerath, Hrvoje Miletic, Eskil Eskilsson, Simone P. Niclou, Morten Lund-Johansen, Gro V. Røsland, Ingrid Moen, Krishna M. Talasila, Olivier Keunen, Lars Prestegarden, Janice M. Nigro, Rolf Bjerkvig, Per Øyvind Enger, Daniel Stieber, Kai Ove Skaftnesmo, Sverre Mørk, Peter C. Huszthy, Jian Wang
Publikováno v:
Acta Neuropathologica
Angiogenesis is regarded as a hallmark of cancer progression and it has been postulated that solid tumor growth depends on angiogenesis. At present, however, it is clear that tumor cell invasion can occur without angiogenesis, a phenomenon that is pa
Autor:
Rupavathana Mahesparan, Daniel Stieber, Hrvoje Miletic, Lars Kaderali, Rolf Bjerkvig, Jian Wang, Manja Schubert, Simone P. Niclou, Evelin Schröck, Andreas von Deimling, Jaime Alberto Campos Valenzuela, Janice M. Nigro, Per Øystein Sakariassen, Terje Sundstrøm, Jörg Balss, Barbara Klink, Anja Torsvik, Peter C. Huszthy, Mads Aarhus
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 3, p e59773 (2013)
PLoS ONE, Vol 8, Iss 3, p e59773 (2013)
Oligodendroglioma poses a biological conundrum for malignant adult human gliomas: it is a tumor type that is universally incurable for patients, and yet, only a few of the human tumors have been established as cell populations in vitro or as intracra
Autor:
Janice M. Nigro, Krishna M. Talasila, Gro V. Røsland, Hrvoje Miletic, Rolf Bjerkvig, Kjersti Mangseth, Simone P. Niclou, Rupavathana Mahesparan, Olav Karsten Vintermyr, Lasse Evensen, Marek Wagner, Anja Torsvik, Daniel Stieber, Narve Brekka
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e81183 (2013)
PLoS ONE
PLoS ONE
Two of the signature genetic events that occur in human gliomas, EGFR amplification and IDH mutation, are poorly represented in experimental models in vitro. EGFR amplification, for example, occurs in 40 to 50% of GBM, and yet, EGFR amplification is