Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Janet D. Vechio"'
Publikováno v:
Cell. 87:447-458
NuMA is a nuclear protein during interphase but redistributes to the spindle poles early in mitosis. To investigate its role during spindle formation, we tested spindle assembly in frog egg extracts from which NuMA was immunodepleted. Immunodepletion
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf478fc46d4fec7a8fcd0660ed33db8b
https://doi.org/10.1016/s0092-8674(00)81365-3
https://doi.org/10.1016/s0092-8674(00)81365-3
Publikováno v:
Annals of Neurology. 40:603-610
Neurofilaments, assembled from NF-L (68 kd), NF-M (95 kd), and NF-H (115 kd), are the most abundant structural components in large myelinated axons, particularly those of motor neurons. Aberrant neurofilament accumulation in cell bodies and axons of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a11554bc8565e73aa21ac8795fb8abc4
https://doi.org/10.1002/ana.410400410
https://doi.org/10.1002/ana.410400410
Autor:
Toni L. Williamson, Lucie Bruijn, Robert H. Brown, Janet D. Vechio, Joseph R. Marszalek, Zuoshang Xu, Michael K. Lee, Don W. Cleveland
Publikováno v:
Cold Spring Harbor Symposia on Quantitative Biology. 61:709-723
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::68ed6c9f3bff6e40ba14e898b1221d0f
https://doi.org/10.1101/sqb.1996.061.01.071
https://doi.org/10.1101/sqb.1996.061.01.071
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 76:1560-1568
Albright hereditary osteodystrophy (AHO) is an inherited disorder associated with deficient activity of the alpha-subunit of the guanine nucleotide-binding regulatory protein (Gs alpha) that couples receptors to adenylyl cyclase. To identify mutation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b5786bae708b2c5b481f08b21fb66b2
https://doi.org/10.1210/jcem.76.6.8388883
https://doi.org/10.1210/jcem.76.6.8388883
Autor:
Joseph R. Marszalek, Don W. Cleveland, Lucie Bruijn, Donald L. Price, X.-S. Xu, Sangram S. Sisodia, Philip C. Wong, Michael K. Lee, David R. Borchelt, Janet D. Vechio
Publikováno v:
Neurology. 47(4 Suppl 2)
To examine the mechanism(s) of disease underlying ALS, transgenic mouse models have been constructed that express aberrant neurofilaments or mutations in the abundant, cytoplasmic enzyme superoxide dismutase 1 (SOD1).In addition to progressive weakne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::34902bd4d568129068e3453220ae27e2
https://pubmed.ncbi.nlm.nih.gov/8858052
https://pubmed.ncbi.nlm.nih.gov/8858052