Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Jana Strakova"'
Publikováno v:
Biomarker Insights, Vol 2010, Iss 5, Pp 119-127 (2010)
Externí odkaz:
https://doaj.org/article/d747dae5a56646409ab7a3187fcec392
Publikováno v:
Biomarker Insights, Vol 5 (2010)
von Willebrand Factor (vWF) is a multimeric plasma protein important for platelet plug formation. As part of its haemostatic role, it is released from endothelial cells during vascular stress or injury and is considered an excellent biomarker of endo
Externí odkaz:
https://doaj.org/article/83f60dedf8ab412bb7f986cf5389ec8a
Autor:
Arnost Vesely, Jana Strakova
Publikováno v:
Comparative Education Review. 63:418-438
Large-scale international assessments, such as PIAAC (Programme for the International Assessment of Adult Competencies) or PISA (Programme for International Student Assessment), have heightened int...
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f725673e5570e95c27b3725a98e180f2
https://doi.org/10.1086/703869
https://doi.org/10.1086/703869
Autor:
James M. Ervasti, Forum Kamdar, Maria Razzoli, Jana Strakova, DeWayne Townsend, Debra Kulhanek, Daniel J. Garry, Alessandro Bartolomucci
Publikováno v:
Scientific Reports, Vol 8, Iss 1, Pp 1-10 (2018)
Scientific Reports
Scientific Reports
Duchenne muscular dystrophy (DMD) is a disease marked by the development of skeletal muscle weakness and wasting. DMD results from mutations in the gene for the cytoskeletal protein dystrophin. The loss of dystrophin expression is not limited to musc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53817dc81202eca41fb2942237a9cdde
http://link.springer.com/article/10.1038/s41598-018-31753-3
http://link.springer.com/article/10.1038/s41598-018-31753-3
Autor:
DeWayne Townsend, Zachary Stelter, Jana Strakova, Katharine M. Sharpe, Kaleb D. Fischer, Amritha Yellamilli
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 310:H938-H948
Duchenne muscular dystrophy (DMD) is a disease of progressive destruction of striated muscle, resulting in muscle weakness with progressive respiratory and cardiac failure. Respiratory and cardiac disease are the leading causes of death in DMD patien
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f43f10821316d90050e39c3c9b70bf7a
https://doi.org/10.1152/ajpheart.00917.2015
https://doi.org/10.1152/ajpheart.00917.2015
Autor:
Katharine M. Sharpe, Jon Dean, Jana Strakova, DeWayne Townsend, Tatyana A. Meyers, Guy L. Odom
Publikováno v:
Journal of Molecular and Cellular Cardiology. 76:106-115
Duchenne muscular dystrophy is a fatal progressive disease of both cardiac and skeletal muscle resulting from the mutations in the DMD gene and loss of the protein dystrophin. Alpha-dystrobrevin (α-DB) tightly associates with dystrophin but the sign
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e81f12e2b715178bbfcb1ba73abc548
https://doi.org/10.1016/j.yjmcc.2014.08.013
https://doi.org/10.1016/j.yjmcc.2014.08.013
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1811:617-625
In the brain, phosphatidylcholine (PC) is synthesized by the CDP-choline pathway in which the rate-limiting step is catalyzed by two isoforms of CTP:phosphocholine cytidylyltransferase (CT): CTα and CTβ2. In mice, CTβ2 mRNA is more highly expresse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b2876c5007abe9e0cc2667cab64bafee
https://doi.org/10.1016/j.bbalip.2011.06.017
https://doi.org/10.1016/j.bbalip.2011.06.017
Autor:
Jiri Jiracek, Lucas Li, Jana Strakova, Kelly T. Williams, Warren D. Kruger, Kevin L. Schalinske, Timothy A. Garrow, Rima Rozen, Sapna Gupta
Publikováno v:
Nutrition Research. 30:492-500
Betaine homocysteine S-methyltransferase (BHMT) catalyzes the transfer of a methyl group from betaine to homocysteine (Hcy), forming dimethylglycine and methionine. We previously showed that inhibiting BHMT in mice by intraperitoneal injection of S-(
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3118940d3a79571b8c728d02d4600539
https://doi.org/10.1016/j.nutres.2010.06.017
https://doi.org/10.1016/j.nutres.2010.06.017
Autor:
Diego Sbrissa, Robert B. Silver, Rajeswari Dondapati, Assia Shisheva, Robert Deeb, Krzysztof Mlak, Ognian C. Ikonomov, Jana Strakova
Publikováno v:
Molecular and Cellular Biology. 24:10437-10447
Multivesicular body morphology and size are controlled in part by PtdIns(3,5)P(2), produced in mammalian cells by PIKfyve-directed phosphorylation of PtdIns(3)P. Here we identify human Vac14 (hVac14), an evolutionarily conserved protein, present in a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6083a23617239bf7b006969774829d08
https://doi.org/10.1128/mcb.24.23.10437-10447.2004
https://doi.org/10.1128/mcb.24.23.10437-10447.2004
Publikováno v:
Endocrinology. 145:4853-4865
The cellular functions and regulation of phosphatidylinositol (PtdIns) 5-phosphate (5-P), the newest addition to the family of phosphoinositides (PIs), are still elusive. Here we have examined a plausible role of PtdIns 5-P as a signaling intermediat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b88e63eb234de613ebd20e8fc3d678eb
https://doi.org/10.1210/en.2004-0489
https://doi.org/10.1210/en.2004-0489