Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Jan-Phillip Stümpel"'
Autor:
Georg Kochs, Beate M. Kümmerer, Isabelle Bekeredjian-Ding, Gunther Hartmann, Evelyn Hartmann, Stephan Herberhold, Christine Schuberth-Wagner, Christoph Coch, Peter Staeheli, Winfried Barchet, Friedrich Bootz, Martin Schlee, Achim Hoerauf, Natalio Garbi, Jan Phillip Stümpel, Vanessa Lilien-Waldau, Janos Ludwig, Dirk Wohlleber
Publikováno v:
Molecular Therapy. 25:2093-2103
Influenza A virus infection causes substantial morbidity and mortality in seasonal epidemic outbreaks, and more efficient treatments are urgently needed. Innate immune sensing of viral nucleic acids stimulates antiviral immunity, including cell-auton
Autor:
Steve Austin, Ben Palmer, P. Batty, Murugaiyan Thanigaikumar, Daniel P. Hart, Jan-Phillip Stümpel, Savita Rangarajan, Carolyn M. Millar, Thynn Thynn Yee, Kate Khair
Publikováno v:
British Journal of Haematology. 176:796-804
Summary Inhibitor formation in non-severe haemophilia A is a life-long risk and associated with morbidity and mortality. There is a paucity of data to understand real-world inhibitor screening practice. We evaluated the treatment burden, haemostatic
Autor:
Evelyn Hartmann, Vanessa von Lilien Waldau, Jan Phillip Stümpel, Janos Ludwig, Martin Schlee, Christoph Coch, Gunther Hartmann
Publikováno v:
The Journal of Immunology. 202:198.12-198.12
Nasal mucosa is a key barrier against upper airway infection including influenza. Trained innate antiviral immunity has the potential to broadly protect the host from a variety of respiratory viruses. Although exposure of cells to type I IFN confers
Autor:
Susann Schulze, Madlen Jentzsch, Nadja Jaekel, Song-Yau Wang, Simone Heyn, Sebastian Schwind, Wolfram Poenisch, Claudia Nehring, Yvonne Remane, Jan-Phillip Stümpel, Sabine Leiblein, Georg-Nikolaus Franke, Vladan Vucinic, Elena Ruschpler, Dietger Niederwieser
Publikováno v:
Blood. 126:5439-5439
Introduction: Up to 30% of patients (pts) undergoing stem cell mobilization with granulocyte colony-stimulating factor (G-CSF) with or without prior chemotherapy mobilize poorly. Plerixafor is a potent CXCR4 receptor antagonist that is licensed for s