Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Jan Hein T. van Waesberghe"'
Autor:
Iris van 't Erve, Nina J. Wesdorp, Jamie E. Medina, Leonardo Ferreira, Alessandro Leal, Joost Huiskens, Karen Bolhuis, Jan-Hein T. van Waesberghe, Rutger-Jan Swijnenburg, Daan van den Broek, Victor E. Velculescu, Geert Kazemier, Cornelis J. Punt, Gerrit A. Meijer, Remond J. Fijneman
Publikováno v:
Cancer Research. 82:519-519
Introduction: The distribution of KRAS mutation variants across tumor types is not uniform. The KRAS A146 mutation is predominantly seen in colorectal cancer (CRC) patients. Here, we evaluated how clinical features like tumor load and overall surviva
Autor:
Saskia Bontemps, Maarten E. Tushuizen, Jan Hein T. van Waesberghe, Petra J. W. Pouwels, Roger K. Schindhelm, Andrea Mari, Mathijs C. Bunck, Robert J. Heine, Michaela Diamant
Publikováno v:
Diabetes Care, 30(11), 2916-2921. American Diabetes Association Inc.
Tushuizen, M E, Bunck, M C, Pouwels, P J, Bontemps, S, Van Waesberghe, J H T, Schindhelm, R K, Mari, A, Heine, R J & Diamant, M 2007, ' Pancreatic fat content and β-cell function in men with and without type 2 diabetes ', Diabetes Care, vol. 30, no. 11, pp. 2916-2921 . https://doi.org/10.2337/dc07-0326
Tushuizen, M E, Bunck, M C, Pouwels, P J, Bontemps, S, Van Waesberghe, J H T, Schindhelm, R K, Mari, A, Heine, R J & Diamant, M 2007, ' Pancreatic fat content and β-cell function in men with and without type 2 diabetes ', Diabetes Care, vol. 30, no. 11, pp. 2916-2921 . https://doi.org/10.2337/dc07-0326
OBJECTIVE—Insulin resistance, associated with increased lipolysis, results in a high exposure of nonadipose tissue to lipids. Experimental data indicate that fatty infiltration of pancreatic islets may also contribute to β-cell dysfunction, but wh
Autor:
Maarten E. Tushuizen, Jan Hein T. van Waesberghe, Mathijs C. Bunck, Robert J. Heine, Petra J. W. Pouwels, Michaela Diamant
Publikováno v:
Tushuizen, M E, Bunck, M C, Pouwels, P J, van Waesberghe, J H T, Diamant, M & Heine, R J 2006, ' Incretin mimetics as a novel therapeutic option for hepatic steatosis ', Liver International, vol. 26, no. 8, pp. 1015-1017 . https://doi.org/10.1111/j.1478-3231.2006.01315.x
Liver International, 26(8), 1015-1017. Wiley-Blackwell
Liver International, 26(8), 1015-1017. Wiley-Blackwell
Background: Fat accumulation in the liver or non-alcoholic fatty liver disease (NAFLD) is regarded as a key pathogenic factor and component of the metabolic syndrome. It was reported that administration of the incretin mimetic exenatide reversed hepa