Zobrazeno 1 - 10
of 157
pro vyhledávání: '"Jan C‐C. Hu"'
Autor:
Mengqi Zhou, Hong Zhang, Heather Camhi, Figen Seymen, Mine Koruyucu, Yelda Kasimoglu, Jung-Wook Kim, Hera Kim-Berman, Ninna M. R. Yuson, Paul J. Benke, Yiqun Wu, Feng Wang, Yaqin Zhu, James P. Simmer, Jan C-C. Hu
Publikováno v:
International Journal of Oral Science, Vol 13, Iss 1, Pp 1-11 (2021)
Abstract Oligodontia is the congenital absence of six or more teeth and comprises the more severe forms of tooth agenesis. Many genes have been implicated in the etiology of tooth agenesis, which is highly variable in its clinical presentation. The p
Externí odkaz:
https://doaj.org/article/b4fae4e706cc42399f6b5e82f45aca92
Autor:
Mengqi Zhou, Hong Zhang, Heather Camhi, Figen Seymen, Mine Koruyucu, Yelda Kasimoglu, Jung-Wook Kim, Hera Kim-Berman, Ninna M. R. Yuson, Paul J. Benke, Yiqun Wu, Feng Wang, Yaqin Zhu, James P. Simmer, Jan C-C. Hu
Publikováno v:
International Journal of Oral Science, Vol 13, Iss 1, Pp 1-1 (2021)
Externí odkaz:
https://doaj.org/article/69aca93b9fdf4733af20c92ca8e7c19f
Autor:
Shih‐Kai Wang, Yuanyuan Hu, Charles E. Smith, Jie Yang, Chunhua Zeng, Jung‐Wook Kim, Jan C‐C. Hu, James P. Simmer
Publikováno v:
Molecular Genetics & Genomic Medicine, Vol 7, Iss 6, Pp n/a-n/a (2019)
Abstract Background Truncation FAM83H mutations cause human autosomal dominant hypocalcified amelogenesis imperfecta (ADHCAI), an inherited disorder characterized by severe hardness defects in dental enamel. No enamel defects were observed in Fam83h
Externí odkaz:
https://doaj.org/article/6b4f5eeb5f434b398802d89035dd84e0
Publikováno v:
Frontiers in Physiology, Vol 8 (2017)
Enamel formation requires consecutive stages of development to achieve its characteristic extreme mineral hardness. Mineralization depends on the initial presence then removal of degraded enamel proteins from the matrix via endocytosis. The ameloblas
Externí odkaz:
https://doaj.org/article/1c720d7d7fcf4635b3fe4b6722ff3d09
Autor:
Shih-Kai Wang, Hong Zhang, Hua-Chieh Lin, Yin-Lin Wang, Shu-Chun Lin, Figen Seymen, Mine Koruyucu, James P. Simmer, Jan C.-C. Hu
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 11, p 6132 (2024)
AMELX mutations cause X-linked amelogenesis imperfecta (AI), known as AI types IE, IIB, and IIC in Witkop’s classification, characterized by hypoplastic (reduced thickness) and/or hypomaturation (reduced hardness) enamel defects. In this study, we
Externí odkaz:
https://doaj.org/article/eda180a328994f63a11ce2a415b4b175
Autor:
Shih‐Kai Wang, Hong Zhang, Yin‐Lin Wang, Hung‐Ying Lin, Figen Seymen, Mine Koruyucu, J. Timothy Wright, Jung‐Wook Kim, James P. Simmer, Jan C.‐C. Hu
Publikováno v:
International Endodontic Journal.
Autor:
Tian Liang, Charles E. Smith, Yuanyuan Hu, Hong Zhang, Chuhua Zhang, Qian Xu, Yongbo Lu, Ling Qi, Jan C.-C. Hu, James P. Simmer
Publikováno v:
Scientific Reports. 13
Dentin sialophosphoprotein (DSPP) is primarily expressed by differentiated odontoblasts (dentin-forming cells), and transiently expressed by presecretory ameloblasts (enamel-forming cells). Disease-causing DSPP mutations predominantly fall into two c
Autor:
Kuan‐Yu Chu, Yin‐Lin Wang, Jung‐Tsu Chen, Chia‐Hui Lin, Chung‐Chen Jane Yao, Yi‐Jane Chen, Huan‐Wen Chen, James P. Simmer, Jan C.‐C. Hu, Shih‐Kai Wang
Publikováno v:
Annals of the New York Academy of Sciences.
Autor:
null Kuan‐Yu Chu, null Yin‐Lin Wang, null Jung‐Tsu Chen, null Chia‐Hui Lin, null Chung‐Chen Jane Yao, null Yi‐Jane Chen, null Huan‐Wen Chen, null James P. Simmer, null Jan C.‐C. Hu, null Shih‐Kai Wang
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::560a01164eb8a938222e5c07c45fb235
https://doi.org/10.1111/nyas.14988/v2/response1
https://doi.org/10.1111/nyas.14988/v2/response1
Autor:
Youn Jung Kim, Hong Zhang, Yejin Lee, Figen Seymen, Mine Koruyucu, Yelda Kasimoglu, James P. Simmer, Jan C.-C. Hu, Jung-Wook Kim
Publikováno v:
Journal of Personalized Medicine
Volume 13
Issue 2
Pages: 326
Volume 13
Issue 2
Pages: 326
Amelogenesis imperfecta (AI) is a heterogeneous collection of hereditary enamel defects. The affected enamel can be classified as hypoplastic, hypomaturation, or hypocalcified in form. A better understanding of normal amelogenesis and improvements in