Zobrazeno 1 - 10
of 232
pro vyhledávání: '"Jamie Fisher"'
Publikováno v:
Judgment and Decision Making, Vol 7, Pp 644-658 (2012)
The current study examines the construct validity of the Maximization Scale (MS; Schwartz et al., 2002) and the Maximization Tendency Scale (MTS; Diab et al., 2008) as well as the nomological net of the maximizing construct. We find that both scales
Externí odkaz:
https://doaj.org/article/1356b46502b94cd19a6194b24f5d9258
Autor:
Sandra Santulli-Marotto, Alexis Gervais, Jamie Fisher, Brandy Strake, Carol Anne Ogden, Chelsea Riveley, Jill Giles-Komar
Publikováno v:
PLoS ONE, Vol 10, Iss 12, p e0145078 (2015)
Defective clearance of apoptotic cells can result in sustained inflammation and subsequent autoimmunity. Macrophages, the "professional phagocyte" of the body, are responsible for efficient, non-phlogistic, apoptotic cell clearance. Controlling phago
Externí odkaz:
https://doaj.org/article/121c70eecd334f97b1f6fa2901ad517d
Publikováno v:
Books. :54-57
Autor:
Jonathan A. Bernstein, Louanne Hudgins, Laurie H. Seaver, Eric Muller, Margaret Homeyer, Susan R. Hintz, Melanie A. Manning, Angela Myers, Edward D. Esplin, Marie-Luise Brennan, Jamie Fisher, Anna-Kaisa Niemi, Cynthia J. Curry
Publikováno v:
American Journal of Medical Genetics Part A. 164:2814-2821
The RASopathies are a family of developmental disorders caused by heritable defects of the RAS/MAPK signaling pathway. While the postnatal presentation of this group of disorders is well known, the prenatal and neonatal findings are less widely recog
Autor:
Ana Coelho, Tadas Panavas, Bingyuan Wu, Theodore Petley, Lester Gutshall, Jamie Fisher, Sandra Santulli-Marotto, Ken Boakye, Michael A. Rycyzyn, Cory M. Hogaboam, Jennifer L. Luongo, Karl Kavalkovich, Mary Ryan
Publikováno v:
Monoclonal Antibodies in Immunodiagnosis and Immunotherapy. 32:162-171
The chemokines CCL17 (TARC) and CCL22 (MDC) function through the same receptor, CCR4, but have been proposed to differentially affect the immune response. To better understand the role of the individual ligands, a panel of rat anti-mouse CCL17 surrog
Autor:
Bernice E. Morrow, Cynthia J. Curry, Robert W. Marion, Cynthia M. Powell, Sheila J. Upton, Elaine Pereira, Eva Andermann, Arthur S. Aylsworth, Abby K. Stevens, Lisa G. Shaffer, Bryce A. Heese, Frederick Andermann, John B. Moeschler, Dina Amrom, Blake C. Ballif, Melissa K. Maisenbacher, Allen N. Lamb, Kandamurugu Manickam, Melanie Babcock, Trilochan Sahoo, Martin Veilleux, Jill A. Rosenfeld, Jay W. Ellison, Cathy A. Stevens, Alex R. Paciorkowski, Wilfredo Torres-Martinez, Jamie Fisher
Publikováno v:
neurogenetics. 13:31-47
Interstitial deletions of 6q are associated with variable phenotypes, including growth retardation, dysmorphic features, upper limb malformations, and Prader-Willi (PW)-like features. Only a minority of cases in the literature have been characterized
Autor:
Jill Giles-Komar, Erin Johnston, Natalie Fursov, Michael A. Rycyzyn, Haiyan Jiang, Gordon Powers, Karen E. Duffy, Jamie Fisher, Theodore Petley, Adam Cotty
Publikováno v:
Hybridoma. 30:153-162
ST2L is a transmembrane receptor that belongs to the IL-1 receptor family. The receptor is expressed on various cell types including Th2 cells, mast cells, basophils, growth-activated fibroblasts, and vascular endothelial cells. ST2L activation by it
Autor:
Karen E. Duffy, Rachel A Bunting, Lena Alexopoulou, Karen A Smalley, Heena Beck, Richard A. Flavell, Ted Petley, Xuesong Liu, Jamie Fisher, Lani San Mateo, Christine K. Ward, Roberta J. Lamb, Holly Raymond
Publikováno v:
Cellular Immunology. 267:9-16
Toll-like receptor 3 (TLR3) binds and signals in response to dsRNA and poly(I:C), a synthetic double stranded RNA analog. Activation of TLR3 triggers innate responses that may play a protective or detrimental role in viral infections or in immune-med
Autor:
A. Micheil Innes, Julien L. Marcadier, Jamie Fisher, Cory Airheart, Elizabeth A. Woods, David A. Dyment, Chandree L. Beaulieu, Wen Qin, Cynthia J. Curry, Alan J. Mears
Publikováno v:
American journal of medical genetics. Part A. (1)
PDAC (also termed Matthew Wood) syndrome is a rare, autosomal recessive disorder characterized by pulmonary hypoplasia/aplasia, diaphragmatic defects, bilateral anophthalmia, and cardiac malformations. The disorder is caused by mutations in STRA6, an
Autor:
Jamie Fisher, Cynthia J. Curry, Rebecca Sahraoui, Claudio Sandoval, Ann Haskins Olney, Jessica X. Chong, Deborah L. Stabley, Katia Sol-Church, Lisa Pilchman, Karen W. Gripp
Patients with physical findings suggestive of Treacher Collins syndrome (TCS) or mandibulofacial dysostosis (MFD) and macrocytic anemia diagnostic of Diamond-Blackfan anemia (DBA) have been reported. Disease-causing genes have been identified for TCS
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da2eec77effa9977e359a55ab9c5572d
https://europepmc.org/articles/PMC4149220/
https://europepmc.org/articles/PMC4149220/