Zobrazeno 1 - 7
of 7
pro vyhledávání: '"James V. Perciaccante"'
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 283:R1362-R1367
We determined whether cerebral arteriolar dilation to N-methyl-d-aspartate (NMDA), a response dependent on stimulation of cortical neurons and inhibited by anoxic stress, would be preserved by hypothermia during and following ischemia. Pial arteriola
Autor:
Zsombor Lacza, James V. Perciaccante, David W. Busija, Bela Kis, Michelle Puskar, Allison W. Miller
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 283:H406-H411
We investigated the mechanism of EDHF-mediated dilation to bradykinin (BK) in piglet pial arteries. Topically applied BK (3 μmol/l) induced vasodilation (62 ± 12%) after the administration of N ω-nitro-l-arginine methyl ester (l-NAME) and indometh
Publikováno v:
Stroke. 30:2713-2719
Background and Purpose — N -Methyl- d -aspartate (NMDA) elicits neuronally mediated cerebral arteriolar vasodilation that is reduced by ischemia/reperfusion (I/R). This sequence has been preserved by pretreatment with the ATP-sensitive potassium (K
Autor:
Thomas M. Louis, Ferenc Domoki, Greg Robins, Roland Veltkamp, Ferenc Bari, James V. Perciaccante, David W. Busija
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 277:H1113-H1118
We have previously shown that cycloheximide (CHX) preserved neuronal function after global cerebral ischemia in piglets, in a manner similar to indomethacin. To elucidate the mechanism of this protection, we tested the hypothesis that CHX would inhib
Autor:
Nishadi Thrikawala, Judy L. Aschner, Gregory S. Robins, Nora Kovacs, David W. Busija, Jorge P. Figueroa, James V. Perciaccante
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 277:H371-H379
We determined the expression and functional correlate of in vitro transfection with a recombinant adenoviral vector encoding the gene for bovine endothelial nitric oxide synthase (AdCMVeNOS) or Escherichia coliβ-galactosidase (AdCMVLacZ) in pulmonar
Autor:
Michelle Puskar, James V. Perciaccante, Katsuyoshi Shimizu, Ferenc Domoki, David W. Busija, Ferenc Bari
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 282(4)
N-methyl-D-aspartate (NMDA) elicits pial arteriolar dilation that has been associated with neuronal nitric oxide (NO) production. However, endothelial factors or glial P-450 epoxygenase products may play a role. We tested whether NMDA-induced pial va
Publikováno v:
Neuroreport. 12(18)
Anoxic stress attenuates NMDA-induced pial arteriolar dilation via a mechanism involving actions of cyclooxygenase (COX)-derived reactive oxygen species (ROS). We examined whether the selective COX-2 inhibitor NS398 would protect neuronal function af