Zobrazeno 1 - 10
of 19
pro vyhledávání: '"James O. Hogan"'
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 324:L259-L270
Severe levels of acidosis (pH < 6.8) have been shown to cause a sustained rise in cytosolic Ca2+ concentration in carotid body Type 1 (glomus) cells. To understand how physiologically relevant levels of acidosis regulate Ca2+ signaling in glomus cell
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a69e1aec23d5e55a48cccc2bd7c93a01
https://doi.org/10.1152/ajplung.00099.2022
https://doi.org/10.1152/ajplung.00099.2022
Publikováno v:
The FASEB Journal. 36
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2de49b03088d131324088c8273558e5b
https://doi.org/10.1096/fasebj.2022.36.s1.r4744
https://doi.org/10.1096/fasebj.2022.36.s1.r4744
Publikováno v:
The FASEB Journal. 36
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9bdada2ee7e2b523dcd23ee3e98c4628
https://doi.org/10.1096/fasebj.2022.36.s1.r2543
https://doi.org/10.1096/fasebj.2022.36.s1.r2543
Publikováno v:
American Journal of Physiology-Cell Physiology. 318:C430-C438
We studied the mechanisms by which carotid body glomus (type 1) cells produce spontaneous Ca2+oscillations in normoxia and hypoxia. In cells perfused with normoxic solution at 37°C, we observed relatively uniform, low-frequency Ca2+oscillations in >
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5137e847f72f9c93aaa6c4ccaee87ba4
https://doi.org/10.1152/ajpcell.00442.2019
https://doi.org/10.1152/ajpcell.00442.2019
Publikováno v:
Respiratory Physiology & Neurobiology. 246:98-106
Cystathionine-γ-lyase (CSE) is a multifunctional enzyme, and hydrogen sulfide (H2S) is one of its products. CSE and H2S have recently been proposed to be critical signaling molecules in hypoxia-induced excitation of carotid body (CB) glomus cells an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bf76773684def2b430a19f0b30f530ac
https://doi.org/10.1016/j.resp.2017.08.009
https://doi.org/10.1016/j.resp.2017.08.009
Publikováno v:
Respiratory Physiology & Neurobiology. 237:13-21
A recent study showed that hypoxia activates a Ca2+-sensitive, Na+-permeable non-selective cation channel (NSC) in carotid body glomus cells. We studied the effects of mitochondrial inhibitors that increase Ca2+ influx via Ca2+ channel (Cav), and rec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::de963d84a179124b760b41c3406526ea
https://doi.org/10.1016/j.resp.2016.12.005
https://doi.org/10.1016/j.resp.2016.12.005
Publikováno v:
Am J Physiol Cell Physiol
We studied the mechanisms by which carotid body glomus (type 1) cells produce spontaneous Ca(2+) oscillations in normoxia and hypoxia. In cells perfused with normoxic solution at 37°C, we observed relatively uniform, low-frequency Ca(2+) oscillation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8414502e200a2b325929763175ea9d39
https://pubmed.ncbi.nlm.nih.gov/31913694
https://pubmed.ncbi.nlm.nih.gov/31913694
Publikováno v:
The FASEB Journal. 33
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::de50645eb32a6f4cd7c0171e324a4f33
https://doi.org/10.1096/fasebj.2019.33.1_supplement.552.2
https://doi.org/10.1096/fasebj.2019.33.1_supplement.552.2
Publikováno v:
The FASEB Journal. 34:1-1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fe83812e146db0bb5eb0793c8e0995a8
https://doi.org/10.1096/fasebj.2020.34.s1.02976
https://doi.org/10.1096/fasebj.2020.34.s1.02976
Publikováno v:
Advances in experimental medicine and biology. 1071
Glomus cells isolated from rabbit and rat/mouse carotid bodies have been used for many years to study the role of ion channels in hypoxia sensing. Studies show that hypoxia inhibits the inactivating K
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c46fd2761edee9a2266af31c4aebdc54
https://pubmed.ncbi.nlm.nih.gov/30357731
https://pubmed.ncbi.nlm.nih.gov/30357731