Zobrazeno 1 - 10
of 65
pro vyhledávání: '"James, Haorah"'
Autor:
Venkatesan Perumal, Arun Reddy Ravula, Agnieszka Agas, Manisha Kannan, Xiangshan Liu, Shanmuga Sundari I, Sivakumar Vijayaraghavalu, James Haorah, Yuanwei Zhang, Namas Chandra
Publikováno v:
Micro, Vol 3, Iss 1, Pp 84-106 (2023)
Traumatic brain injury (TBI) is a major cause of morbidity and mortality worldwide, affecting over 10 million people annually, with an estimated cost of $76.5 billion. Although apocynin freely transverses the blood–brain barrier (BBB), its applicat
Externí odkaz:
https://doaj.org/article/1fcc9f4797174370813177656aa7e0a5
Autor:
Xiaotang Ma, Agnieszka Agas, Zain Siddiqui, KaKyung Kim, Patricia Iglesias-Montoro, Jagathi Kalluru, Vivek Kumar, James Haorah
Publikováno v:
Bioactive Materials, Vol 5, Iss 1, Pp 124-132 (2020)
Traumatic brain injury (TBI) impacts over 3.17 million Americans. Management of hemorrhage and coagulation caused by vascular disruption after TBI is critical for the recovery of patients. Cerebrovascular pathologies play an important role in the und
Externí odkaz:
https://doaj.org/article/0be257f4c18041a39ecf5211d3f980d9
Autor:
Harisritha Kataru, Jagathi Kalluru, James Haorah, Brooke Leiser, Ricardo Garcia, Agnieszka Agas
Publikováno v:
Alcohol. 94:25-41
Alcohol use and HIV-1 infection have a pervasive impact on brain function, which extends to the requirement, distribution, and utilization of energy within the central nervous system. This effect on neuroenergetics may explain, in part, the exacerbat
Publikováno v:
Alcohol. 86:65-74
Engagement of programmed death-1 (PD-1) receptor by its ligands (PD-L1/PD-L2) in activated immune cells is known to be involved in inflammatory neurological disease via a co-inhibitory signal pathway. Interaction of PD-1/PD-L1 is believed to occur on
Autor:
Agnieszka Agas, Jagathi Kalluru, James Haorah, Zain Siddiqui, Patricia Iglesias-Montoro, Vivek A. Kumar, Ka Kyung Kim, Xiaotang Ma
Publikováno v:
Bioactive Materials
Bioactive Materials, Vol 5, Iss 1, Pp 124-132 (2020)
Bioactive Materials, Vol 5, Iss 1, Pp 124-132 (2020)
Traumatic brain injury (TBI) impacts over 3.17 million Americans. Management of hemorrhage and coagulation caused by vascular disruption after TBI is critical for the recovery of patients. Cerebrovascular pathologies play an important role in the und
Autor:
Yiming Cheng, Kevin D. Belfield, Arun Reddy Ravula, Xiaotang Ma, James Haorah, Agnieszka Agas
Publikováno v:
SSRN Electronic Journal.
Hemorrhage is often a major component of traumatic brain injury (TBI). Red blood cells (RBCs) accumulated at the hemorrhagic site undergo hemolysis upon energy depletion. Hemolysis of RBCs is expected to release free iron into the central nervous sys
Publikováno v:
Journal of Neuroimmune Pharmacology. 15:181-195
Traumatic brain injury (TBI) is a major health problem for over 3.17 million people in the US, attracting increasing public attentions. Understanding the underlying mechanism of TBI is urgent for better diagnosis and treatment. Here, we examined the
Publikováno v:
Journal of Neuroimmunology. 368:577869
The trans-activator of transcription (TAT) is a human immunodeficiency virus (HIV-1) regulatory protein that is actively sloughed by infected cells. Once released, TAT can injure bystander cells and bring about their dysfunction. In the presence of e
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e70833 (2013)
Neuropathy and neurocognitive deficits are common among chronic alcohol users, which are believed to be associated with mitochondrial dysfunction in the brain. The specific type of brain mitochondrial respiratory chain complexes (mRCC) that are adver
Externí odkaz:
https://doaj.org/article/aba616c481b24a2e9ecd46d1e1be00e7
Publikováno v:
PLoS ONE, Vol 6, Iss 4, p e19258 (2011)
Methamphetamine (METH), an addictive psycho-stimulant drug exerts euphoric effects on users and abusers. It is also known to cause cognitive impairment and neurotoxicity. Here, we hypothesized that METH exposure impairs the glucose uptake and metabol
Externí odkaz:
https://doaj.org/article/bc3ae6f402f84d7f83f460f3f694bfa7