Zobrazeno 1 - 10
of 388
pro vyhledávání: '"Jalil, E."'
Publikováno v:
In International Journal of Refrigeration February 2022 134:312-319
Publikováno v:
Journal of Applied Toxicology 21(5): 403-407 (2001)
Some markers of oxidative injury were measured in different rat brain areas (hippocampus, cerebral cortex, striatum, hypothalamus, amygdala/piriform cortex and cerebellum) after the systemic administration of an excitotoxic dose of kainic acid (KA, 9
Externí odkaz:
http://arxiv.org/abs/0708.2061
Publikováno v:
European Journal of Pharmacology 390(3): 295-298 (2000)
Kainate induces a marked expression of cyclooxygenase-2 after its systemic administration. Because cyclooxygenase-2 activity is associated to the production of reactive oxygen species, we investigated the effects of nimesulide, a selective cyclooxyge
Externí odkaz:
http://arxiv.org/abs/0708.1971
Publikováno v:
Brain Research 990(1-2): 1-7 (2003)
Recent experimental evidences indicate that pyruvate, the final metabolite of glycolysis, has a remarkable protective effect against different types of brain injury. The purpose of this study was to assess the neuroprotective effect and the neurologi
Externí odkaz:
http://arxiv.org/abs/0708.1781
Autor:
Candelario-Jalil, E., Alvarez, D., Castaneda, J. M., Al-Dalain, S. M., Martinez-Sanchez, G., Merino, N., Leon, O. S.
Publikováno v:
Brain Research 927(2): 212-215 (2002)
Several studies suggest that cyclooxygenase-2 contributes to the delayed progression of ischemic brain damage. In this study we examined whether the highly selective cyclooxygenase-2 inhibitor DFU reduces neuronal damage when administered several hou
Externí odkaz:
http://arxiv.org/abs/0708.0581
Autor:
Candelario-Jalil, E., Alvarez, D., Gonzalez-Falcon, A., Garcia-Cabrera, M., Martinez-Sanchez, G., Merino, N., Giuliani, A., Leon, O. S.
Publikováno v:
European Journal of Pharmacology 453(2-3): 189-195 (2002)
Cyclooxygenase-2 is involved in the inflammatory component of the ischemic cascade, playing an important role in the delayed progression of the brain damage. The present study evaluated the pharmacological effects of the selective cyclooxygenase-2 in
Externí odkaz:
http://arxiv.org/abs/0708.0564
Autor:
Candelario-Jalil, E., Akundi, R. S., Bhatia, H. S., Lieb, K., Appel, K., Munoz, E., Hull, M., Fiebich, B. L.
Publikováno v:
Journal of Neuroimmunology 174(1-2): 39-51 (2006)
In the present study, we show that ascorbic acid dose-dependently inhibited interleukin-1beta (IL-1beta)-mediated PGE2 synthesis in the human neuronal cell line, SK-N-SH. Furthermore, in combination with aspirin, ascorbic acid augmented the inhibitor
Externí odkaz:
http://arxiv.org/abs/0708.0548
Publikováno v:
Neuroscience Research 41(3): 233-241 (2001)
The time course of oxidative damage in different brain regions was investigated in the gerbil model of transient cerebral ischemia. Animals were subjected to both common carotid arteries occlusion for 5 min. After the end of ischemia and at different
Externí odkaz:
http://arxiv.org/abs/0708.0572
Publikováno v:
Neuroscience Research 47(2): 245-253 (2003)
Metabolism of arachidonic acid by cyclooxygenase is one of the primary sources of reactive oxygen species in the ischemic brain. Neuronal overexpression of cyclooxygenase-2 has recently been shown to contribute to neurodegeneration following ischemic
Externí odkaz:
http://arxiv.org/abs/0708.0559