Zobrazeno 1 - 10
of 67
pro vyhledávání: '"Jaime Herrera-Acosta"'
Autor:
Richard J. Johnson, Bernardo Rodriguez-Iturbe, Takahiko Nakagawa, Duk-Hee Kang, Dan I. Feig, Jaime Herrera-Acosta
Publikováno v:
Hypertension.
Autor:
Laura G. Sánchez-Lozada, Pablo Bautista, Jaime Herrera-Acosta, Virgilia Soto, Tomás Nepomuceno, Adriana Jiménez, Martha Franco, Carmen Avila-Casado, Richard J. Johnson, Magdalena Cristóbal, Takahiko Nakagawa, Edilia Tapia
Publikováno v:
American Journal of Physiology-Renal Physiology. 292:F423-F429
Fructose intake has been recently linked to the epidemic of metabolic syndrome and, in turn, the metabolic syndrome has been epidemiologically linked with renal progression. The renal hemodynamic effects of fructose intake are unknown, as well as the
Autor:
Bernardo Rodriguez-Iturbe, José Santamaría, Jaime Herrera-Acosta, Rocio Bautista, Tomás Nepomuceno, Martha Franco, Edilia Tapia, Richard J. Johnson, Flavio Martínez, Angélica Montoya
Publikováno v:
American Journal of Physiology-Renal Physiology. 291:F1281-F1287
Transient administration of ANG II causes persistent salt-sensitive hypertension associated with arteriolopathy, interstitial inflammation, and cortical vasoconstriction; blocking the vascular and inflammatory changes with mycophenolate mofetil (MMF)
Autor:
Katherine R. Tuttle, Robert Short, Jaime Herrera-Acosta, Richard J. Johnson, Hanbo Hu, Sergey Zharikov, Daniel I. Feig, Xiaosen Ouyang, Takahiko Nakagawa, Jawaharlal M. Patel, Olena Glushakova, Edward R. Block
Publikováno v:
American Journal of Physiology-Renal Physiology. 290:F625-F631
The worldwide epidemic of metabolic syndrome correlates with an elevation in serum uric acid as well as a marked increase in total fructose intake (in the form of table sugar and high-fructose corn syrup). Fructose raises uric acid, and the latter in
Autor:
Richard J. Johnson, L. Gabriela Sánchez-Lozada, Jaime Herrera-Acosta, Marilda Mazzali, Takahiko Nakagawa, Duk Hee Kang
Publikováno v:
Blood Purification. 24:67-70
Uric acid might often be regarded as a simple marker of renal disease. Although it is well known that hyperuricemia causes gout which is associated with renal insufficiency and cardiovascular disease, one might think that it could attribute to the in
Publikováno v:
Kidney International. 68:S82-S86
Tubulointerstitial damage and progression of renal failure . The present work reviews the mechanisms and close association between glomerular and tubular damage and its relationship to renal functional impairment. In addition, we present an overview
Autor:
Carlos Roncal, Christine Gersch, Thomas M. Coffman, Richard J. Johnson, Jaime Herrera-Acosta, Bernardo Rodriguez-Iturbe, Xiaosen Ouyang, Wei Mu, Thu H. Le
Publikováno v:
American Journal of Physiology-Renal Physiology. 289:F902-F910
AT1 double receptor (AT1A and AT1B) knockout mice have lower blood pressure, impaired growth, and develop early renal microvascular disease and tubulointerstitial injury. We hypothesized that there would be an increased expression of vasoactive, prof
Autor:
Bernardo Rodriguez-Iturbe, Richard J. Johnson, Jaime Herrera-Acosta, Daniel I. Feig, Duk Hee Kang
Publikováno v:
American Journal of Hypertension. 18:431-440
We present the hypothesis that most cases of essential hypertension occur via two phases. The first phase is initiated by episodes of renal vasoconstriction induced by a hyperactive sympathetic nervous system, activation of the renin-angiotensin syst
Autor:
Bernardo Rodriguez-Iturbe, Duk Hee Kang, Jaime Herrera-Acosta, Takahiko Nakagawa, Daniel I. Feig, Richard J. Johnson
Publikováno v:
Hypertension. 45:326-330
Despite the fact that essential hypertension was originally described as hypertension occurring in the absence of clinical renal disease,1 Dahl et al later showed by transplantation studies in experimental models of hypertension that the kidney is ul
Autor:
Virgilia Soto, Edilia Tapia, Jaime Herrera-Acosta, Bernardo Rodriguez-Iturbe, Tomás Nepomuceno, Carmen Avila-Casado, José Santamaría, Laura G. Sánchez-Lozada, Richard J. Johnson
Publikováno v:
Kidney International. 67:237-247
Mild hyperuricemia induces vasoconstriction and maintains glomerular hypertension in normal and remnant kidney rats.BackgroundHyperuricemia has been associated with renal disease. Because glomerular hemodynamic alterations critically contribute to in