Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Jai Seong Park"'
Autor:
Jong-Uk Lee, Jong-Sook Park, Eunjeong Seo, Jin Seol Kim, Hae Ung Lee, Yongjin Chang, Jai Seong Park, Choon-Sik Park
Publikováno v:
BMC Pulmonary Medicine, Vol 24, Iss 1, Pp 1-8 (2024)
Abstract Purpose The extent of honeycombing and reticulation predict the clinical prognosis of IPF. Emphysema, consolidation, and ground glass opacity are visible in HRCT scans. To date, there have been few comprehensive studies that have used these
Externí odkaz:
https://doaj.org/article/cf6109a28d104647b8f22e784a02c59f
Autor:
Ji-Hye Son, Jong-Uk Lee, Susie Chin, Eun-Suk Go, Jai-Seong Park, Hwa-Kyun Shin, Hun Soo Chang, Jong-Sook Park, Choon-Sik Park
Publikováno v:
The Korean Journal of Internal Medicine, Vol 36, Iss 4, Pp 914-923 (2021)
Background/Aims Receptor tyrosine kinase-like orphan receptor 2 (ROR2) is a major regulator of Wnt signaling, which is involved in fibroblast dysfunction. Because its role has not been evaluated in idiopathic pulmonary fibrosis (IPF), we examined the
Externí odkaz:
https://doaj.org/article/8650283cca794a47a23f688cea7f2ab0
Autor:
Choon-Sik Park, Jong-Sook Park, Jai-Seong Park, Hun Soo Chang, Ji-Hye Son, Jong-Uk Lee, Susie Chin, Eun-Suk Go, Hwa-Kyun Shin
Publikováno v:
The Korean Journal of Internal Medicine
The Korean Journal of Internal Medicine, Vol 36, Iss 4, Pp 914-923 (2021)
The Korean Journal of Internal Medicine, Vol 36, Iss 4, Pp 914-923 (2021)
Background/Aims Receptor tyrosine kinase-like orphan receptor 2 (ROR2) is a major regulator of Wnt signaling, which is involved in fibroblast dysfunction. Because its role has not been evaluated in idiopathic pulmonary fibrosis (IPF), we examined the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fdaabbd83f52be87941b622cfdece278
http://europepmc.org/articles/PMC8273837
http://europepmc.org/articles/PMC8273837
Autor:
Myung Shin Kim, Choon-Sik Park, Eun-Suk Go, Jong-Uk Lee, Jai-Seong Park, Hun Soo Chang, Jong-Sook Park
Publikováno v:
B34. ILD CELLULAR MECHANISMS.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e418f96641e9f69b6fb0a75cd48dd373
https://doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3083
https://doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3083
Autor:
Jong-Uk Lee, Jong-Sook Park, Myung-Shin Kim, Jai-Seong Park, Eun-Suk Go, Hun Soo Chang, Choon-Sik Park
Background: Neutrophilic inflammation is a predominant characteristic of idiopathic pulmonary fibrosis (IPF). S100 Calcium-Binding Protein A9 (S100A9) is a neutrophil-derived protein and is involved in the development of neutrophil-related chronic in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5048dce3cf78d7e0a99bccbf48b6f128
https://doi.org/10.21203/rs.3.rs-20772/v1
https://doi.org/10.21203/rs.3.rs-20772/v1
Autor:
Jong Uk Lee, Eun-Young Shim, Eun-Suk Koh, Jai-Seong Park, Choon-Sik Park, Hun Soo Chang, Jong-Sook Park, Hwa-Kyun Shin
Publikováno v:
Respiratory Medicine. 171:105945
Fibroblast dysfunction is the main pathogenic mechanism of idiopathic pulmonary fibrosis (IPF). S100 calcium-binding protein A4 (S100A4) plays critical roles in the proliferation of fibroblasts and in the development of pulmonary, hepatic, and renal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f1165293ae35c2697004407c7bf6a47
https://doi.org/10.1016/j.rmed.2020.105945
https://doi.org/10.1016/j.rmed.2020.105945