Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Jacqueline S. Beesley"'
Autor:
Jacqueline S. Beesley, Douglas A. Coulter, Bryan Kavanaugh, Akiva S. Cohen, Takayuki Itoh, Judith B. Grinspan, Aki Itoh, David E Pleasure
Publikováno v:
Journal of Neurochemistry. 81:390-402
Cells of the oligodendroglial lineage express Ca2+-permeable alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate-preferring glutamate receptors (AMPA-GluR) during development. Prolonged activation of their AMPA-GluR causes Ca2+ overload, resulting i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3b7420997931ddd0a50d53b1808edbec
https://doi.org/10.1046/j.1471-4159.2002.00866.x
https://doi.org/10.1046/j.1471-4159.2002.00866.x
Publikováno v:
Journal of Neuroscience Research. 66:497-505
As oligodendrocytes mature they progress through a series of distinct differentiation steps characterized by the expression of specific markers. One such marker, polysialic acid found on the neural cell adhesion molecule (NCAM), is detected by antibo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6fddaed175f9d69e34eca890b4063bc9
https://doi.org/10.1002/jnr.10002
https://doi.org/10.1002/jnr.10002
Autor:
Theodore Xu, Abdolmohammad Rostami, Marina Scarlato, Peter Bannerman, Usha R. Reddy, David E Pleasure, Steven S. Scherer, Jacqueline S. Beesley
Publikováno v:
Journal of Neuroscience Research. 66:16-22
We screened for genes whose expression is significantly up- or downregulated during Wallerian degeneration in adult rat sciatic nerve with cDNA arrays. Fibroblast growth factor-5 (FGF-5) mRNA seemed to be induced. This was confirmed by northern blott
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::12de21d670fcf5e88cc56f81856e5636
https://doi.org/10.1002/jnr.1193
https://doi.org/10.1002/jnr.1193
Autor:
Franca Cambi, Judith B. Grinspan, John Kamholz, Huibin Yang, Huiyuan Jiang, Raj Awatramani, Jacqueline S. Beesley, James Y. Garbern
Publikováno v:
Journal of Neuroscience Research. 61:376-387
Myelin, a multilamellar membrane structure that facilitates nerve conduction, is synthesized in the central nervous system (CNS) by oligodendrocytes. Gtx, a member of the homeodomain family of transcriptional factors, is a candidate regulator of myel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::23fbdee02bc9a69625600c81ab33b196
https://doi.org/10.1002/1097-4547(20000815)61:4<376::aid-jnr4>3.0.co
https://doi.org/10.1002/1097-4547(20000815)61:4<376::aid-jnr4>3.0.co
Publikováno v:
Journal of Neuroscience Research. 59:430-435
We used cDNA arrays to investigate molecular aspects of the differentiation of an immortalized line of oligodendroglial progenitors, and of immunopan-purified primary cultures of oligodendroglial progenitors, to immature oligodendroglia. Developmenta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9a1d209ebd6f9da1dd08857dd45fa93b
https://doi.org/10.1002/(sici)1097-4547(20000201)59:3<430::aid-jnr18>3.0.co
https://doi.org/10.1002/(sici)1097-4547(20000201)59:3<430::aid-jnr18>3.0.co
Publikováno v:
Journal of Cellular Biochemistry. 76:270-279
Oligodendrocyte differentiation is accompanied by dramatic changes in gene expression as well as cell cycle arrest. To determine whether cell cycle arrest is sufficient to induce the changes in cell phenotype associated with differentiation, we inhib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c64652409db648b81d1b53ea688f4a36
https://doi.org/10.1002/(sici)1097-4644(20000201)76:2<270::aid-jcb10>3.0.co
https://doi.org/10.1002/(sici)1097-4644(20000201)76:2<270::aid-jcb10>3.0.co
Autor:
Aki Itoh, Bryan Kavanaugh, Judith B. Grinspan, Jacqueline S. Beesley, Takayuki Itoh, David E Pleasure
Publikováno v:
Journal of Neuroscience Research. 60:725-732
Prior reports demonstrated that cells of the oligodendroglial lineage are susceptible to excitotoxic necrosis mediated by alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic acid glutamate receptors (AMPA-GluR), and also showed that these cells expre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e3d66ff72a8b2b4a2a97e56757d059b3
https://doi.org/10.1002/1097-4547(20000615)60:6<725::aid-jnr4>3.0.co
https://doi.org/10.1002/1097-4547(20000615)60:6<725::aid-jnr4>3.0.co
Autor:
Yoko Yamaya, David E Pleasure, Akira Yoshioka, Shinji Saiki, Masumi Kanemoto, Jacqueline S. Beesley, Genjiro Hirose
Publikováno v:
Brain Research. 854:207-215
Cells of oligodendroglial lineage are susceptible to oxygen and glucose deprivation. When oligodendrocyte-like cells differentiated from CG-4-immortalized rat O-2A progenitor cells were exposed to hypoxia alone or glucose deprivation alone for 48 h,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c116fb5a2a7d318829db6941413c6659
https://doi.org/10.1016/s0006-8993(99)02359-8
https://doi.org/10.1016/s0006-8993(99)02359-8
Autor:
David F. Carpio, Steven S. Scherer, Jacqueline S. Beesley, Judith B. Grinspan, Markella Coulalaglou
Publikováno v:
Journal of Neuroscience Research. 54:623-634
Mutations in the proteolipid protein gene (PLP/plp), which encodes the major intrinsic membrane protein in central nervous system (CNS) myelin, cause inherited dysmyelination in mammals. One of these mutants, the myelin-deficient (md) rat, has severe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cec40a6426cfbe8f25a9ee39f036acdb
https://doi.org/10.1002/(sici)1097-4547(19981201)54:5<623::aid-jnr7>3.0.co
https://doi.org/10.1002/(sici)1097-4547(19981201)54:5<623::aid-jnr7>3.0.co
Publikováno v:
Journal of neuroscience research. 64(4)
The myelin-deficient (MD) rat has a point mutation in its proteolipid protein (PLP) gene that causes severe dysmyelination and oligodendrocyte cell death. Using an in vitro model, we have shown that MD oligodendrocytes initially differentiate similar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b7c476bcec2558b176234fc7f785015
https://pubmed.ncbi.nlm.nih.gov/11340644
https://pubmed.ncbi.nlm.nih.gov/11340644