Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Jacqueline Gayet"'
Publikováno v:
Cell reports. 41(2)
Midget and parasol ganglion cells (GCs) represent the major output channels from the primate eye to the brain. On-type midget and parasol GCs exhibit a higher background spike rate and thus can respond more linearly to contrast changes than their Off
Adapting between scotopic and photopic illumination involves switching the routing of retinal signals between rod and cone-dominated circuits. In the daytime, cone signals pass through parallel On and Off cone bipolar cells, that are sensitive to inc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3acf5b48b23efe70806c846fa9cfadba
https://doi.org/10.1101/2020.05.17.100982
https://doi.org/10.1101/2020.05.17.100982
Publikováno v:
The Journal of Neuroscience. 38:3414-3427
Silent voltage-gated potassium channel subunits (KVS) interact selectively with members of the KV2 channel family to modify their functional properties. The localization and functional roles of these silent subunits remain poorly understood. Mutation
Autor:
Lauren Renner, Xiaojie Wang, Trevor J. McGill, Lois M. A. Colgin, Samuel M. Peterson, Brandy L. Dozier, Jacqueline Gayet, Cassandra Cullin, Jonathan Stoddard, Kamm Prongay, Martha Neuringer, Betsy Ferguson, Anne D. Lewis, Teresa Puthussery
Publikováno v:
Exp Eye Res
The development of therapies for retinal disorders is hampered by a lack of appropriate animal models. Higher nonhuman primates are the only animals with retinal structure similar to humans, including the presence of a macula and fovea. However, few
Publikováno v:
eNeuro
Adapting between scotopic and photopic illumination involves switching the routing of retinal signals between rod and cone-dominated circuits. In the daytime, cone signals pass through parallel On and Off cone bipolar cells (CBCs), that are sensitive
Publikováno v:
Gayet-Primo, J; & Puthussery, T. (2015). Alterations in Kainate Receptor and TRPM1 Localization in Bipolar Cells after Retinal Photoreceptor Degeneration. FRONTIERS IN CELLULAR NEUROSCIENCE, 9. doi: 10.3389/fnce1.2015.00436. UC Berkeley: Retrieved from: http://www.escholarship.org/uc/item/00s5m33t
Frontiers in cellular neuroscience, vol 9, iss DEC
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 9 (2015)
Frontiers in cellular neuroscience, vol 9, iss DEC
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 9 (2015)
Photoreceptor degeneration differentially impacts glutamatergic signaling in downstream On and Off bipolar cells. In rodent models, photoreceptor degeneration leads to loss of glutamatergic signaling in On bipolar cells, whereas Off bipolar cells app
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f0b308ab67e5e1ae5b9d80971ecf820
http://www.escholarship.org/uc/item/00s5m33t
http://www.escholarship.org/uc/item/00s5m33t
Publikováno v:
European Journal of Neuroscience. 34:1419-1431
Mutation of the gene encoding carbonic anhydrase-related protein VIII (CAVIII) results in motor coordination deficits in mice and humans, due to loss of this protein in Purkinje cells of the cerebellum. Recent studies have indicated that the CAVIII g
Publikováno v:
The Journal of comparative neurology, vol 518, iss 4
Secretagogin, a recently cloned member of the EF-hand family of calcium binding proteins, was localized in the mouse, rat, and rabbit retina using immunofluorescence immunohistochemistry. Secretagogin is expressed in sub-populations of ON and OFF con
Publikováno v:
The Journal of Comparative Neurology. 501:931-943
In the brain and the retina metabotropic glutamate receptors (mGluRs) modulate synaptic transmission; in particular, L-2-amino-4-phosphonobutyrate-sensitive group-III mGluRs are generally presynaptic and provide negative feedback of neurotransmitter
Autor:
Robert M. Duvoisin, Salma Quraishi, Connie Zhang, Jacqueline Gayet-Primo, Timothy Pfankuch, Heather O'Connor, Jacob Raber
Publikováno v:
European Journal of Neuroscience. 22:425-436
To study the role of the metabotropic glutamate receptor 8 (mGluR8), mice lacking this receptor were generated by homologous recombination. Homozygous mGluR8-deficient mice are about 8% heavier than their wild-type age-matched controls after reaching