Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Jacob Zimbric"'
Autor:
Rhiannon E. Jones, Sehyun Oh, Julia W. Grimstead, Jacob Zimbric, Laureline Roger, Nicole H. Heppel, Kevin E. Ashelford, Kate Liddiard, Eric A. Hendrickson, Duncan M. Baird
Publikováno v:
Cell Reports, Vol 8, Iss 4, Pp 1063-1076 (2014)
Short dysfunctional telomeres are capable of fusion, generating dicentric chromosomes and initiating breakage-fusion-bridge cycles. Cells that escape the ensuing cellular crisis exhibit large-scale genomic rearrangements that drive clonal evolution a
Externí odkaz:
https://doaj.org/article/74ca282653444cada7d066278696395b
Autor:
Adam J. Harvey, Sehyun Oh, Eric A. Hendrickson, Pauline Jackson, Yongbao Wang, Thanh Nguyen, Jacob Zimbric
Publikováno v:
DNA Repair. 21:97-110
Ku-dependent C-NHEJ (classic non-homologous end joining) is the primary DNA EJing (end joining) repair pathway in mammals. Recently, an additional EJing repair pathway (A-NHEJ; alternative-NHEJ) has been described. Currently, the mechanism of A-NHEJ
Publikováno v:
Nucleic Acids Research
Classic non-homologous end joining (C-NHEJ) is the predominant DNA double-strand break repair pathway in humans. Although seven genes Ku70, Ku86, DNA-PK(cs), Artemis, DNA Ligase IV (LIGIV), X-ray cross-complementing group 4 and XRCC4-like factor are
Autor:
Julia W. Grimstead, Jacob Zimbric, Nicole H. Heppel, Duncan M. Baird, Kate Liddiard, Sehyun Oh, Eric A. Hendrickson, Laureline Roger, Rhiannon E. Jones, Kevin E. Ashelford
Publikováno v:
Cell Reports
Cell Reports, Elsevier Inc, 2014, 8 (4), pp.1063-1076. ⟨10.1016/j.celrep.2014.07.007⟩
Cell Reports, Vol 8, Iss 4, Pp 1063-1076 (2014)
Cell Reports, 2014, 8 (4), pp.1063-1076. ⟨10.1016/j.celrep.2014.07.007⟩
Cell Reports, Elsevier Inc, 2014, 8 (4), pp.1063-1076. ⟨10.1016/j.celrep.2014.07.007⟩
Cell Reports, Vol 8, Iss 4, Pp 1063-1076 (2014)
Cell Reports, 2014, 8 (4), pp.1063-1076. ⟨10.1016/j.celrep.2014.07.007⟩
SummaryShort dysfunctional telomeres are capable of fusion, generating dicentric chromosomes and initiating breakage-fusion-bridge cycles. Cells that escape the ensuing cellular crisis exhibit large-scale genomic rearrangements that drive clonal evol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::20c64dfa69f72f64f951085ba311f7e9
https://hal.archives-ouvertes.fr/hal-03204504
https://hal.archives-ouvertes.fr/hal-03204504