Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Jacob P. S. Johnson"'
Autor:
Kelly J. Gregory, Amy L. Roberts, Erin M. Conlon, Jacob A. Mayfield, Mary J. Hagen, Giovanna M. Crisi, Brooke A. Bentley, Jeffrey J. Kane, Grace Makari-Judson, Holly S. Mason, Jun Yu, Lihua Julie Zhu, Karl Simin, Jacob P. S. Johnson, Ashraf Khan, Ben R. Schneider, Sallie S. Schneider, D. Joseph Jerry
Publikováno v:
Breast Cancer Research, Vol 21, Iss 1, Pp 1-18 (2019)
Abstract Background Atypical breast hyperplasias (AH) have a 10-year risk of progression to invasive cancer estimated at 4–7%, with the overall risk of developing breast cancer increased by ~ 4-fold. AH lesions are estrogen receptor alpha positive
Externí odkaz:
https://doaj.org/article/14367b64f7854897a6a4e3a757b9c8bf
Autor:
Jack L Leonard, Deborah M Leonard, Scot A Wolfe, Jilin Liu, Jaime Rivera, Michelle Yang, Ryan T Leonard, Jacob P S Johnson, Prashant Kumar, Kate L Liebmann, Amanda A Tutto, Zhongming Mou, Karl J Simin
Publikováno v:
PLoS ONE, Vol 12, Iss 9, p e0184458 (2017)
[This corrects the article DOI: 10.1371/journal.pone.0181724.].
Externí odkaz:
https://doaj.org/article/bbf9eb4f25cb452ba73c52962bc98659
Autor:
Jack L Leonard, Deborah M Leonard, Scot A Wolfe, Jilin Liu, Jaime Rivera, Michelle Yang, Ryan T Leonard, Jacob P S Johnson, Prashant Kumar, Kate L Liebmann, Amanda A Tutto, Zhongming Mou, Karl J Simin
Publikováno v:
PLoS ONE, Vol 12, Iss 7, p e0181724 (2017)
Members of the Dickkopf (Dkk) family of Wnt antagonists interrupt Wnt-induced receptor assembly and participate in axial patterning and cell fate determination. One family member, DKK3, does not block Wnt receptor activation. Loss of Dkk3 expression
Externí odkaz:
https://doaj.org/article/314b504f6fa84f28abb31e68d7b35583
Publikováno v:
Current treatment options in psychiatry. 9(3)
Exposure to potentially morally injurious events (PMIEs) and the development of moral injury have yet to be conceptualized as they relate to social determinants of health (SDoH).Recent Findings.In this paper, the extant literature on moral injury and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fdaa71f5bba21df49fd594859de00f32
https://pubmed.ncbi.nlm.nih.gov/35756696
https://pubmed.ncbi.nlm.nih.gov/35756696
Autor:
Prashant Kumar, Malini Mukherjee, Jacob P S Johnson, Milan Patel, Bing Huey, Donna G Albertson, Karl Simin
Publikováno v:
PLoS Genetics, Vol 8, Iss 11, p e1003027 (2012)
Breast cancers that are "triple-negative" for the clinical markers ESR1, PGR, and HER2 typically belong to the Basal-like molecular subtype. Defective Rb, p53, and Brca1 pathways are each associated with triple-negative and Basal-like subtypes. Our m
Externí odkaz:
https://doaj.org/article/42087c78485c4195b3ae59a180aa529d
Autor:
Jacob P. S. Johnson, D. Joseph Jerry, Ben R. Schneider, Karl Simin, Giovanna M. Crisi, Sallie S. Schneider, Ashraf Khan, Lihua Julie Zhu, Jacob A. Mayfield, Holly Mason, Kelly J. Gregory, Mary J. Hagen, Erin M. Conlon, Grace Makari-Judson, Jeffrey J. Kane, Amy L. Roberts, Brooke A. Bentley, Jun Yu
Publikováno v:
Breast Cancer Research : BCR
Breast Cancer Research, Vol 21, Iss 1, Pp 1-18 (2019)
Breast Cancer Research, Vol 21, Iss 1, Pp 1-18 (2019)
Background Atypical breast hyperplasias (AH) have a 10-year risk of progression to invasive cancer estimated at 4–7%, with the overall risk of developing breast cancer increased by ~ 4-fold. AH lesions are estrogen receptor alpha positive (ERα+) a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::de3636010783c94a3f173a722352f152
https://pubmed.ncbi.nlm.nih.gov/31248446
https://pubmed.ncbi.nlm.nih.gov/31248446
Autor:
Kate L. Liebmann, Jacob P. S. Johnson, Deborah M. Leonard, Michelle Yang, Karl Simin, Zhongming Mou, Amanda A. Tutto, Jack L. Leonard, Prashant Kumar, Scot A. Wolfe, Jilin Liu, Ryan T. Leonard, Jaime J. Rivera
Publikováno v:
PLoS ONE
PLoS ONE, Vol 12, Iss 7, p e0181724 (2017)
PLoS ONE, Vol 12, Iss 7, p e0181724 (2017)
Members of the Dickkopf (Dkk) family of Wnt antagonists interrupt Wnt-induced receptor assembly and participate in axial patterning and cell fate determination. One family member, DKK3, does not block Wnt receptor activation. Loss of Dkk3 expression
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::22cb89975f131644ee23e292cf7c8fb6
https://pubmed.ncbi.nlm.nih.gov/28863194
https://pubmed.ncbi.nlm.nih.gov/28863194
Autor:
Amanda A. Tutto, Jacob P. S. Johnson, Zhongming Mou, Jack L. Leonard, Michelle Yang, Kate L. Liebmann, Ryan T. Leonard, Karl Simin, Scot A. Wolfe, Jilin Liu, Prashant Kumar, Deborah M. Leonard, Jaime J. Rivera
Publikováno v:
PLoS ONE, Vol 12, Iss 9, p e0184458 (2017)
[This corrects the article DOI: 10.1371/journal.pone.0181724.].
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a243fdfa01f054a3620e17bbb4373ba
https://doi.org/10.1371/journal.pone.0184458
https://doi.org/10.1371/journal.pone.0184458
Publikováno v:
Cancer genomicsproteomics. 11(3)
We previously developed a mouse model of breast cancer that mimics human triple-negative breast cancer (TNBC) by inactivating the Retinoblastoma (Rb), Transformation related protein 53 (p53), and Breast cancer 1 (Brca1) pathways in the mammary gland.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::3b75592f5c529f88aa80be79c9aabb04
https://pubmed.ncbi.nlm.nih.gov/24969692
https://pubmed.ncbi.nlm.nih.gov/24969692
Autor:
Jacob P. S. Johnson, Prashant Kumar, Malini Mukherjee, Milan K. Patel, Karl Simin, Bing Huey, Donna G. Albertson
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 8, Iss 11, p e1003027 (2012)
PLoS Genetics, Vol 8, Iss 11, p e1003027 (2012)
Breast cancers that are “triple-negative” for the clinical markers ESR1, PGR, and HER2 typically belong to the Basal-like molecular subtype. Defective Rb, p53, and Brca1 pathways are each associated with triple-negative and Basal-like subtypes. O
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::309bd9b76ccf057c6abd4e3e67ec1fd9
https://pubmed.ncbi.nlm.nih.gov/23173005
https://pubmed.ncbi.nlm.nih.gov/23173005