β-Cyclodextrins alter the energy metabolism-related enzyme activities in rats

Autor: FRANCINE R. IANISKI, AMANDA L. DE OLIVEIRA, NATHANA JAMILLE MEZZOMO, ITIANE D. DE FRANCESCHI, GUILHERME M. DO CARMO, CAMILA R. CREMONESE, MATHEUS D. BALDISSERA, JENIFER P. ZANON, JENIFER KOLLING, JORDANA DANIELA FRIEDERICH, IVANA Z. SILVA, JANICE L. GIONGO, LUCIANE R. FEKSA, RODRIGO A. VAUCHER, CLOVIS MILTON D. WANNMACHER, VIRGINIA C. RECH

Publikováno v: Anais da Academia Brasileira de Ciências, Vol 95, Iss 2 (2023)

Abstract Although widely used in medicine, separation technology, and other fields, the effects of cyclodextrins on the activities of phosphoryl transfer enzymes have not been previously evaluated. In vivo studies evaluated the function of cyclodextr

Externí odkaz: https://doaj.org/article/0d7f80d5450f493ab9eecafd4d2829de

Resveratrol and resveratrol-hydroxypropyl-β-cyclodextrin complex recovered the changes of creatine kinase and Na+, K+-ATPase activities found in the spleen from streptozotocin-induced diabetic rats

Autor: Janice L. Giongo, Luciane Rosa Feksa, Janaína Kolling, Rodrigo de Almeida Vaucher, Matheus D. Baldissera, Nathana J. Mezzomo, Guilherme M. do Carmo, Angela T. S. Wyse, Itiane Diehl de Franceschi, Vivian S.K. Nishihira, Jenifer Kolling, Clovis Milton Duval Wannmacher, Virginia Cielo Rech, Liana da Silva Fernandes, Cláudia Grigolo Pinto, Gilberto Orengo

Publikováno v: Anais da Academia Brasileira de Ciências v.91 n.3 2019
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Volume: 91, Issue: 3, Article number: e20181330, Published: 09 SEP 2019
Anais da Academia Brasileira de Ciências, Vol 91, Iss 3

Type 1 diabetes (T1D) is the result of the selective destruction of the pancreatic β-cells by T cells of the immune system. Although spleen is a secondary lymphoid organ, it is also involved in the T1D pathogenesis. However, the alterations in a var

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ec3c3853d5c8bd3fe9bbe8b5da0d79f1
https://pubmed.ncbi.nlm.nih.gov/31508665