Zobrazeno 1 - 9
of 9
pro vyhledávání: '"J. Y. H. Chan"'
Publikováno v:
European Journal of Clinical Investigation. 38:812-819
Background The effects of cyclooxygenase-1 (COX1) and cyclooxygenase-2 (COX2) inhibition on insulin resistance in subjects with the metabolic syndrome remain elusive. Aims of this study were to examine the effects of COX1 and COX2 inhibitors on whole
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::84ed75d44007063530882e66bb997408
https://doi.org/10.1111/j.1365-2362.2008.02026.x
https://doi.org/10.1111/j.1365-2362.2008.02026.x
Publikováno v:
European Journal of Clinical Investigation. 38:640-648
BACKGROUND Portal endotoxaemia has been speculated to be crucially involved in the pathogenesis of chronic hepatic inflammation, which is highly associated with the development of type 2 diabetes mellitus. This study tests whether portal endotoxaemia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::277d4b27a0c2987b4e1f06f7b47a32a9
https://doi.org/10.1111/j.1365-2362.2008.01991.x
https://doi.org/10.1111/j.1365-2362.2008.01991.x
Autor:
Anthony E. James, Q. Li, Nelson L.S. Tang, Ben Chung-Lap Chan, Paul B.S. Lai, K.-N. Lai, Pak Leong Lim, S. Chen, J. Y.-H. Chan, Yiu-Loon Chui, Pang-Chui Shaw, J. C.-K. Leung, K. K.-H. Lee, Arthur K.K. Ching, Ka Fai To
Publikováno v:
Oncogene. 27:1208-1217
BRE binds to the cytoplasmic domains of tumor necrosis factor receptor-1 and Fas, and in cell lines can attenuate death receptor-initiated apoptosis by inhibiting t-BID-induced activation of the mitochondrial apoptotic pathway. Overexpression of BRE
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23deabf9fb6e0606da77825cf7e7b83d
https://doi.org/10.1038/sj.onc.1210733
https://doi.org/10.1038/sj.onc.1210733
Autor:
Guillermina Lozano, S. T. Liu, S. L. Wang, Yi-Lin Chiu, C. L. Ho, S. M. Huang, Ying-Chuan Chen, J. Y H Chan
Publikováno v:
Cell death and differentiation. 20(5)
The transcription factor p53 is a multifunctional tumor suppressor that arrests the cell cycle in response to stress and modulates the DNA repair process or induces apoptosis. The cellular level and activity of p53 are tightly controlled to maintain
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4be7ddc37c5b44eb1577cb18a3b2964c
https://pubmed.ncbi.nlm.nih.gov/23370271
https://pubmed.ncbi.nlm.nih.gov/23370271
Publikováno v:
Biochemical and Biophysical Research Communications. 206:764-774
We have identified and sequenced a new gene from human cells that is responsive to DNA damage and retinoic acid treatment, and it is highly expressed in brain and reproductive organs (BRE). This BRE gene encodes an mRNA of 1.7-1.9 kb, with an open re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8325527eee4869c1c9f954811bee2213
https://doi.org/10.1006/bbrc.1995.1108
https://doi.org/10.1006/bbrc.1995.1108
Publikováno v:
European journal of clinical investigation. 38(11)
The effects of cyclooxygenase-1 (COX1) and cyclooxygenase-2 (COX2) inhibition on insulin resistance in subjects with the metabolic syndrome remain elusive. Aims of this study were to examine the effects of COX1 and COX2 inhibitors on whole body and m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8140c1a5a14285f7d97e1219d5ed3bfa
https://pubmed.ncbi.nlm.nih.gov/19021698
https://pubmed.ncbi.nlm.nih.gov/19021698
Publikováno v:
Biochemical and biophysical research communications. 191(3)
An inhibitor for DNA ligase I has recently been purified from human cells. This inhibitor of 55-75 kDa forms a reversible complex with DNA ligase I, but has no effect on DNA ligase II and T4 DNA ligase, suggesting that it may play a regulatory role f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1428f8e5dbb344af392a60c3a4e6918
https://pubmed.ncbi.nlm.nih.gov/8466479
https://pubmed.ncbi.nlm.nih.gov/8466479
Publikováno v:
Journal of Endocrinology; Jun2005, Vol. 185 Issue 3, p507-517, 11p
Autor:
F. F. Becker, J. Y.-H. Chan
Publikováno v:
Journal of Biological Chemistry. 263:18231-18235
Cells from patients with Bloom's syndrome (BS), an autosomal recessive disorder associated with an increased risk of cancer, exhibit genomic instability. Increased numbers of sister-chromatid exchanges (SCE) and delayed DNA chain maturation are typic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7b1bee304659b36b1ddce76f48222d5d
https://doi.org/10.1016/s0021-9258(19)81350-9
https://doi.org/10.1016/s0021-9258(19)81350-9