Zobrazeno 1 - 10
of 32
pro vyhledávání: '"J. TUOKKO"'
Publikováno v:
Biomaterial Investigations in Dentistry, Vol 9, Iss 1, Pp 33-40 (2022)
Objective The aim was to investigate the effect of different interfacial surface treatments on the shear bond strength (SBS) between a short fiber-reinforced flowable composite (SFRC) and a particulate-filled flowable composite (PFC). In addition, SB
Externí odkaz:
https://doaj.org/article/6d4633848c0d4766b5f3f834a2acfda6
Autor:
U Yli-Kerttula, Markku Hakala, Jorma Ilonen, J. Tuokko, Susanna Laivoranta-Nyman, Auli Toivanen, Timo Möttönen, Pekka Hannonen, Reijo Luukkainen
Publikováno v:
Annals of the Rheumatic Diseases. 59:173-177
OBJECTIVE To search for possible immunogenetic differencies between the patients with familial and non-familial rheumatoid arthritis (RA). METHODS The study compared 129 familial RA patients with 217 non-familial patients for the frequencies of HLA-D
Publikováno v:
Scandinavian Journal of Rheumatology. 27:225-229
Sixty patients with reactive arthritis (ReA) and 40 with rheumatoid arthritis (RA), were typed for H LA-B27 and class II antigens DR and DQ, and studied for TAP2 gene polymorphism in comparison with 60 healthy controls. TAP2 polymorphisms at position
Autor:
H. REIJONEN, S. NEJENTSEV, J. TUOKKO, S. KOSKINEN, E. TUOMILEHTO-WOLF, H. K. AKERBLOM, J. ILONEN, THE CHILDHOOD DIABETES IN FINLAND S GROUP
Publikováno v:
European Journal of Immunogenetics. 24:357-363
We determined the distribution of DR4 subtypes in 309 DQB1*0302-positive haplotypes found in insulin-dependent diabetes mellitus (IDDM) patients and 70 control haplotypes present only in healthy family members. An increased frequency of DRB1*0401 all
Autor:
Jorma Ilonen, Kaisa Anttila, Timo Möttönen, J. Tuokko, Auli Toivanen, U Yli-Kerttula, Helena Reijonen, S Nikkari
Publikováno v:
Annals of the Rheumatic Diseases. 56:37-40
OBJECTIVE To study HLA class II association in reactive arthritis. METHODS 63 patients with reactive arth-ritis and 46 with rheumatoid arthritis were included in the study. HLA-DR alleles were determined by using a sequence specific PCR method. Oligo
Publikováno v:
Arthritis and rheumatism. 44(2)
Objective To further characterize the HLA gene products that play an important role in the pathogenesis of rheumatoid arthritis (RA). Methods One hundred thirty-four haplotypes from 67 Finnish RA patients and 77 control haplotypes were analyzed for H
Publikováno v:
British journal of rheumatology. 37(11)
The purpose was to study tumour necrosis factor (TNF)-a, -b and -c microsatellites as potential new susceptibility markers for reactive arthritis (ReA). Fifty-nine patients typed for HLA-B27 were studied for frequencies of TNF microsatellite alleles
Autor:
H, Reijonen, S, Nejentsev, J, Tuokko, S, Koskinen, E, Tuomilehto-Wolf, H K, Akerblom, J, Ilonen
Publikováno v:
European journal of immunogenetics : official journal of the British Society for Histocompatibility and Immunogenetics. 24(5)
We determined the distribution of DR4 subtypes in 309 DQB1*0302-positive haplotypes found in insulin-dependent diabetes mellitus (IDDM) patients and 70 control haplotypes present only in healthy family members. An increased frequency of DRB1*0401 all
Publikováno v:
Clinical and experimental rheumatology. 13(5)
The purpose of the present work was to find out whether there is an HLA type common to the patients who, in spite of being B27 negative, have developed reactive arthritis (ReA). We compared the HLA-antigens of 25 HLA-B27 negative ReA patients to thos
Autor:
A. Pinto, S. Adams, K. Ahring, H. Allen, M.F. Almeida, D. Garcia-Arenas, N. Arslan, M. Assoun, Y. Atik Altınok, D. Barrio-Carreras, A. Belanger Quintana, S.M. Bernabei, C. Bontemps, F. Boyle, G. Bruni, M. Bueno-Delgado, G. Caine, R. Carvalho, A. Chrobot, K. Chyż, B. Cochrane, C. Correia, K. Corthouts, A. Daly, S. De Leo, A. Desloovere, A. De Meyer, A. De Theux, B. Didycz, M.E. Dijsselhof, K. Dokoupil, J. Drabik, C. Dunlop, W. Eberle-Pelloth, K. Eftring, J. Ekengren, I. Errekalde, S. Evans, A. Foucart, L. Fokkema, L. François, M. French, E. Forssell, C. Gingell, C. Gonçalves, H. Gökmen Özel, A. Grimsley, G. Gugelmo, E. Gyüre, C. Heller, R. Hensler, I. Jardim, C. Joost, M. Jörg-Streller, C. Jouault, A. Jung, M. Kanthe, N. Koç, I.L. Kok, T. Kozanoğlu, B. Kumru, F. Lang, K. Lang, I. Liegeois, A. Liguori, R. Lilje, O. Ļubina, P. Manta-Vogli, D. Mayr, C. Meneses, C. Newby, U. Meyer, S. Mexia, C. Nicol, U. Och, S.M. Olivas, C. Pedrón-Giner, R. Pereira, K. Plutowska-Hoffmann, J. Purves, A. Re Dionigi, K. Reinson, M. Robert, L. Robertson, J.C. Rocha, C. Rohde, S. Rosenbaum-Fabian, A. Rossi, M. Ruiz, J. Saligova, A. Gutiérrez-Sánchez, A. Schlune, K. Schulpis, J. Serrano-Nieto, A. Skarpalezou, R. Skeath, A. Slabbert, K. Straczek, M. Giżewska, A. Terry, R. Thom, A. Tooke, J. Tuokkola, E. van Dam, T.A.M. van den Hurk, E.M.C. van der Ploeg, K. Vande Kerckhove, M. Van Driessche, A.M.J. van Wegberg, K. van Wyk, C. Vasconcelos, V. Velez García, J. Wildgoose, T. Winkler, J. Żółkowska, J. Zuvadelli, A. MacDonald
Publikováno v:
Molecular Genetics and Metabolism Reports, Vol 18, Iss , Pp 39-44 (2019)
Background: In phenylketonuria (PKU), weaning is considered more challenging when compared to feeding healthy infants. The primary aim of weaning is to gradually replace natural protein from breast milk or standard infant formula with solids containi
Externí odkaz:
https://doaj.org/article/357846d0650c409a859338dfc2ebb4fc