Zobrazeno 1 - 7
of 7
pro vyhledávání: '"J. S. Sprouse"'
The hypothalamic suprachiasmatic nucleus (SCN) is a circadian oscillator that functions as a biological clock. SCN circadian activity is modulated by serotonergic input arising from the median raphe nucleus. The effects of this monoamine neurotransmi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a28cc11430deece1088407f1dc68e74
https://doi.org/10.1016/b978-0-12-809324-5.02391-9
https://doi.org/10.1016/b978-0-12-809324-5.02391-9
Autor:
J. S. Sprouse, L. O. Wilkinson
Publikováno v:
International Review of Psychiatry. 7:5-15
SummarySerotonin 1A receptors are being targeted by a variety of drugs, including par & agonists, full agonists, antagonists. Some serotonin 1A receptors are presynaptic autoreceptors, and others are postsynaptic. Drug actions on serotonin IA recepto
Publikováno v:
Advances in experimental medicine and biology. 294
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 204(1)
The objectives of this study were to investigate 1) the tissue distribution of procainamide acetylase activity in the rat and 2) the kinetics of procainamide acetylation by rat liver and kidney N-acetyltransferase and 3) to determine the effect of dr
Autor:
J S Sprouse, George K. Aghajanian
Publikováno v:
Neuropharmacology. 27(7)
In low cerveau isole transected rats, the effects of microiontophoretic application of putative serotonin 5-HT1A and 5-HT1B agonists on the spontaneous firing rate of CA1 pyramidal cells were compared to those of 5-HT. In contrast to the large curren
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 207(3)
The effects of isoniazid (INH), hydralazine, salicylazosulfapyridine, and sulfapyridine on the quantitative disposition of procainamide (PA) in the intact rat were examined. A dose-dependent inhibition of PA acetylation was observed after coadministr
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 235(3)
Neostigmine-induced muscle fasciculations, quantitated as fasciculatory muscle action potentials, served as an indirect in vivo indicator of motor nerve ending (MNE) excitability. By this method, MNE excitability was depressed in adrenalectomized rat