Zobrazeno 1 - 7
of 7
pro vyhledávání: '"J. P. Noveral"'
Autor:
J. P. Noveral, Michael M. Grunstein
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 269:L339-L343
The tachykinins, substance P (SP) and neurokinins A (NKA) and B (NKB), have been identified in the respiratory tract and implicated in mediating neurogenic inflammation of the airways. To the extent that these neuropeptides may be involved in the pat
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 267:L761-L765
Insulin-like growth factors (IGFs) mediate cell proliferation and differentiation and bind with high affinities and specificities to IGF receptors and IGF-binding proteins (IGFBPs). We examined the roles of these three groups of proteins in cultured
Autor:
J. P. Noveral, Michael M. Grunstein
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 267:L291-L299
To evaluate the potential role of adrenergic receptors in regulating airway smooth muscle (ASM) cell proliferation, the mitogenic effects and mechanisms of action of selective alpha- and beta-adrenoceptor activation were investigated in cultured rabb
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 263:L317-L324
Increased expression of the potent vasoconstrictor and bronchoactive peptide, endothelin-1 (ET-1), has recently been demonstrated in airway epithelial and endothelial cells of asthmatic patients. To identify its potential role in contributing to airw
Publikováno v:
The American journal of physiology. 269(2 Pt 1)
The insulin-like growth factor (IGF) axis is involved in regulating proliferation in a variety of cell types, including airway smooth muscle. Because airway hyperplasia is a characteristic feature of asthma and other lung diseases, we examined the in
Autor:
Michael M. Grunstein, J. P. Noveral
Publikováno v:
The American journal of physiology. 263(5 Pt 1)
Thromboxane (Tx)A2 has been reported to play an important role in modulating airway contractility under various conditions associated with airways inflammation. To identify its potential role in contributing to airway smooth muscle (ASM) hyperplasia,
Publikováno v:
Advances in experimental medicine and biology. 172