Zobrazeno 1 - 10
of 46
pro vyhledávání: '"J. Matuskova"'
Publikováno v:
Journal of Nanomedicine & Nanotechnology.
Autor:
Kristina Krajcirovicova, Václav Pelouch, Michaela Adamcova, Fedor Simko, Olga Pechanova, Ludovit Paulis, J. Matuskova
Publikováno v:
International Journal of Cardiology. 190:128-130
a Department of Pathophysiology, School of Medicine, Comenius University, Bratislava, Slovak Republic b 3rd Clinic of Internal Medicine, School of Medicine, Comenius University, Bratislava, Slovak Republic c Institute of Experimental Endocrinology, S
Autor:
Michaela Adamcova, A. Potacova, Kristina Krajcirovicova, Václav Pelouch, Fedor Simko, Olga Pechanova, Ludovit Paulis, J. Matuskova, J. Simko, Ivan Hulin, Pavol Janega
Publikováno v:
Acta Physiologica. 194:45-55
Aim: We investigated, whether the substrate for nitric oxide (NO) formation –l-arginine – and the aldosterone receptor antagonist – spironolactone – are able to reverse alterations of the left ventricle (LV) and aorta in Nω-nitro-l-arginine
Autor:
J. Matuskova, Michaela Adamcova, Fedor Simko, Olga Pechanova, Ludovit Paulis, V Pelouch, A. Potacova, Kristina Krajcirovicova
Publikováno v:
ResearcherID
Scopus-Elsevier
Scopus-Elsevier
N(G)-nitro-L-arginine-methyl ester (L-NAME)-induced hypertension is associated with protein remodeling of the left ventricle. The aim of the study was to show, whether aldosterone receptor blocker spironolactone and precursor of NO-production L-argin
Publikováno v:
Kidney International. 70(1):170-176
Although angiotensin-converting enzyme (ACE) inhibitors are well-established drugs in the treatment of hypertension, they are not supposed to be sufficient in the inhibition of aldosterone formation. The present study analyzes the effect of aldostero
Autor:
Ivan Hulin, J. Matuskova, Pavel Babal, Fedor Simko, Ivan Luptak, Iveta Bernatova, Olga Pechanova
Publikováno v:
Annals of the New York Academy of Sciences. 967:454-462
AIM The hereditary hypertriglyceridemic (hHTg) rat is characterized by insulin resistance, hypertension, and hypertriglyceridemia. Thus, we investigated whether (a) remodeling of the heart left ventricle (LV) is present under the given hypertensive s
Publikováno v:
Annals of the New York Academy of Sciences. 967:469-475
OBJECTIVE Hereditary hypertriglyceridemia (hHTG) in rats was found to be associated with metabolic abnormalities and elevation of blood pressure. There is controversy regarding the relation between hHTG and vascular function. The aim of this study wa
Autor:
Jarmila Kucharská, Pavel Babal, Iveta Bernatova, Fedor Simko, J. Matuskova, J. Simko, Olga Pechanova, Z. Sumbalova, A. Gvozdjakova, K. Krajcirovicova, Ivan Luptak
Publikováno v:
European Journal of Clinical Investigation. 35:362-368
Background We investigated whether the substrate for nitric oxide synthesis l-arginine is able to modify hypertension and left ventricular hypertrophy development induced by chronic blockade of nitric oxide synthase activity by NG-nitro-l-arginine-me
Autor:
Kristina Krajcirovicova, Olga Pechanova, Ivan Luptak, J. Matuskova, Anna Gvozdjáková, Fedor Simko, Pavel Babal, Jarmila Kucharská
Publikováno v:
Life Sciences. 74:1211-1224
3-Hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors have been shown to prevent or reverse hypertrophy of the LV in several models of left ventricular hypertrophy. The aim of the present study was to determine whether treatment with s
Autor:
Ivan Luptak, Stvrtina S, Fedor Simko, Pomsár J, Olga Pechanova, Pelouch, Ludovit Paulis, Pincíková T, J. Matuskova, Kristina Krajcirovicova
Publikováno v:
Scopus-Elsevier
Europe PubMed Central
Europe PubMed Central
Aldosterone receptor antagonist, spironolactone, has been shown to prevent remodeling of the heart in several models of left ventricular hypertrophy. The aim of the present study was to determine whether the treatment with spironolactone can prevent