Zobrazeno 1 - 10
of 23
pro vyhledávání: '"J. Martínez-Quiroz"'
Autor:
D. Hernández-Martínez, L. Rocha, J. Martínez-Quiroz, C. Morgado-Valle, J. Manzo, M.L. López-Meraz
Publikováno v:
Neurología, Vol 33, Iss 9, Pp 577-582 (2018)
Resumen: Introducción: El status epilepticus (SE) es un tipo de actividad epiléptica que causa atrofia cerebelar y pérdida de células de Purkinje en humanos y en animales de experimentación. El cerebelo es una región con alto contenido de ácid
Externí odkaz:
https://doaj.org/article/97abb21b771346179ff512983d5618f3
Autor:
D. Hernández-Martínez, L. Rocha, J. Martínez-Quiroz, C. Morgado-Valle, J. Manzo, M.L. López-Meraz
Publikováno v:
Neurología (English Edition), Vol 33, Iss 9, Pp 577-582 (2018)
Introduction: Status epilepticus (SE) is an epileptic condition that can cause cerebellar atrophy and loss of Purkinje cells in both humans and research animals. Cerebellum is a region rich in γ-amino butyric acid (GABA) and glutamate, and some stud
Externí odkaz:
https://doaj.org/article/867e501b8da740579ba37599c59352ac
Autor:
C. Rincón-López, A. Tlapa-Pale, J.-S. Medel-Matus, J. Martínez-Quiroz, J.F. Rodríguez-Landa, M.-L. López-Meraz
Publikováno v:
Neurología (English Edition), Vol 32, Iss 9, Pp 587-594 (2017)
Background: Interleukin-1β (IL-1β) increases necrotic neuronal cell death in the CA1 area after induced status epilepticus (SE) in developing rats. However, it remains uncertain whether IL-1β has a similar effect on the hippocampal dentate gyrus (
Externí odkaz:
https://doaj.org/article/501cdb134ec14930a5c266e6030dd01e
Autor:
C. Rincón-López, A. Tlapa-Pale, J.-S. Medel-Matus, J. Martínez-Quiroz, J.F. Rodríguez-Landa, M.-L. López-Meraz
Publikováno v:
Neurología, Vol 32, Iss 9, Pp 587-594 (2017)
Resumen: Introducción: La interleucina 1β (IL-1β) aumenta la muerte neuronal necrótica debido al estado epiléptico (EE) en el área CA1 del hipocampo de ratas en desarrollo; sin embargo, se desconoce si ejerce un efecto similar en el giro dentad
Externí odkaz:
https://doaj.org/article/9ede6ff0e6994ee79f8e2330455a8272
Autor:
M.L. López-Meraz, J. Martínez-Quiroz, D. Hernández-Martínez, Jorge Manzo, Luisa Rocha, Consuelo Morgado-Valle
Publikováno v:
Neurología, Vol 33, Iss 9, Pp 577-582 (2018)
Resumen: Introducción: El status epilepticus (SE) es un tipo de actividad epiléptica que causa atrofia cerebelar y pérdida de células de Purkinje en humanos y en animales de experimentación. El cerebelo es una región con alto contenido de ácid
Autor:
Maria-Leonor Lopez-Meraz, Jesús-Servando Medel-Matus, Juan Francisco Rodríguez-Landa, C. Rincón-López, J. Martínez-Quiroz, A. Tlapa-Pale
Publikováno v:
Neurología (English Edition), Vol 32, Iss 9, Pp 587-594 (2017)
Background: Interleukin-1β (IL-1β) increases necrotic neuronal cell death in the CA1 area after induced status epilepticus (SE) in developing rats. However, it remains uncertain whether IL-1β has a similar effect on the hippocampal dentate gyrus (
Autor:
D, Hernández-Martínez, L, Rocha, J, Martínez-Quiroz, C, Morgado-Valle, J, Manzo, M L, López-Meraz
Publikováno v:
Neurologia (Barcelona, Spain). 33(9)
Status epilepticus (SE) is an epileptic condition that can cause cerebellar atrophy and loss of Purkinje cells in both humans and research animals. Cerebellum is a region rich in γ-aminobutyric acid (GABA) and glutamate, and some studies have shown
Autor:
A. Tlapa-Pale, J. Martínez-Quiroz, Maria-Leonor Lopez-Meraz, Juan Francisco Rodríguez-Landa, C. Rincón-López, Jesús S Medel-Matus
Publikováno v:
Neurología, Vol 32, Iss 9, Pp 587-594 (2017)
Resumen: Introducción: La interleucina 1β (IL-1β) aumenta la muerte neuronal necrótica debido al estado epiléptico (EE) en el área CA1 del hipocampo de ratas en desarrollo; sin embargo, se desconoce si ejerce un efecto similar en el giro dentad
Autor:
Luis Beltran-Parrazal, Dulce-Mariely Álvarez-Croda, Maria-Leonor Lopez-Meraz, Ángel Alberto Puig-Lagunes, J. Martínez-Quiroz, Jesús S Medel-Matus, Juan Santiago-García
Publikováno v:
Instituto Politécnico Nacional
IPN
Redalyc-IPN
Anais da Academia Brasileira de Ciências (Brasil) Num.1 Vol.88
Anais da Academia Brasileira de Ciências, Volume: 88 Supplement 1, Pages: 653-663, Published: 10 MAY 2016
Anais da Academia Brasileira de Ciências v.88 suppl.1 2016
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Vol 88, Iss suppl 1, Pp 653-663 (2016)
IPN
Redalyc-IPN
Anais da Academia Brasileira de Ciências (Brasil) Num.1 Vol.88
Anais da Academia Brasileira de Ciências, Volume: 88 Supplement 1, Pages: 653-663, Published: 10 MAY 2016
Anais da Academia Brasileira de Ciências v.88 suppl.1 2016
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Vol 88, Iss suppl 1, Pp 653-663 (2016)
The contribution of Interleukin-1β (IL-1β) to neuronal injury induced by status epilepticus (SE) in the immature brain remains unclear. The goal of this study was to determine the hippocampal expression of IL-1β and its type 1 receptor (IL-1RI) fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ff2d9a32346a0c9484b8eafad0c1eece
https://www.redalyc.org/articulo.oa?id=32746362020
https://www.redalyc.org/articulo.oa?id=32746362020
Autor:
J. Martínez-Quiroz, Dulce-Mariely Álvarez-Croda, Luis Beltran-Parrazal, Maria-Leonor Lopez-Meraz, Jesús-Servando Medel-Matus, Consuelo Morgado-Valle
Publikováno v:
International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. 38
Interleukin-1β (IL-1β) is associated with seizure-induced neuronal cell death in the adult brain. The contribution of IL-1β to neuronal injury induced by status epilepticus (SE) in the immature brain remains unclear. In the present study, we inves