Zobrazeno 1 - 10
of 216
pro vyhledávání: '"J. M. Brotchie"'
Publikováno v:
Parkinsonismrelated disorders. 78
Long-term treatment of Parkinson's disease (PD) with l-DOPA typically leads to development of l-DOPA induced dyskinesia (LID). Amantadine, an NMDA antagonist, attenuates LID, but with limited efficacy and considerable side-effects. NLX-112 (also know
Autor:
Qi, Li‐Feng‐Rong1 (AUTHOR), Liu, Shuai1,2 (AUTHOR), Fang, Qiuyuan3 (AUTHOR), Qian, Cheng1 (AUTHOR), Peng, Chao4,5 (AUTHOR), Liu, Yuci1 (AUTHOR), Yang, Peng1 (AUTHOR), Wu, Ping4,5 (AUTHOR), Shan, Ling6 (AUTHOR), Cui, Qinghua7 (AUTHOR), Hua, Qian8 (AUTHOR), Yang, Sen9 (AUTHOR), Ye, Cunqi9 (AUTHOR), Yang, Wei2 (AUTHOR) yangwei@zju.edu.cn, Li, Ping1 (AUTHOR) 1019880888@cpu.edu.cn, Xu, Xiaojun1,2 (AUTHOR) xiaojunxu@zju.edu.cn
Publikováno v:
Advanced Science. 10/23/2024, Vol. 11 Issue 39, p1-22. 22p.
Autor:
Cheng, Yan‐Qiong1 (AUTHOR), Zhang, Ruo‐Xi1 (AUTHOR), Li, Xing‐Yuan1 (AUTHOR), Zhou, Xiao‐Ting1 (AUTHOR), Chen, Ming2 (AUTHOR), Liu, Ai‐Jun1 (AUTHOR) mrliuaijun@163.com
Publikováno v:
CNS Neuroscience & Therapeutics. Oct2024, Vol. 30 Issue 10, p1-11. 11p.
Publikováno v:
Neuropharmacology (2006).
info:cnr-pdr/source/autori:J. Lee, V. Di Marzo and J. M. Brotchie/titolo:Role for vanilloid receptor 1 (TRPV1) and endocannabinoid signalling in the regulation of spontaneous and L-DOPA induced locomotion in normal and reserpine-treated rats/doi:/rivista:Neuropharmacology/anno:2006/pagina_da:/pagina_a:/intervallo_pagine:/volume
info:cnr-pdr/source/autori:J. Lee, V. Di Marzo and J. M. Brotchie/titolo:Role for vanilloid receptor 1 (TRPV1) and endocannabinoid signalling in the regulation of spontaneous and L-DOPA induced locomotion in normal and reserpine-treated rats/doi:/rivista:Neuropharmacology/anno:2006/pagina_da:/pagina_a:/intervallo_pagine:/volume
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=cnr_________::42b6b24f724d84c8cd37335013f553c4
http://www.cnr.it/prodotto/i/15327
http://www.cnr.it/prodotto/i/15327
Publikováno v:
Drug newsperspectives. 13(5)
Current symptomatic treatments for Parkinson's disease are based largely on dopamine replacement therapies; however, the fact that these treatments are characterized by many long-term side effects has led to widespread interest in nondopaminergic the
Publikováno v:
Advances in neurology. 91
Autor:
S H, Fox, J M, Brotchie
Publikováno v:
Movement disorders : official journal of the Movement Disorder Society. 15(6)
The involvement of abnormalities in nondopaminergic transmitter systems in Parkinson's disease is noteworthy because of the complications, such as dyskinesia, associated with long-term dopamine replacement therapy. The output regions of the basal gan
Autor:
J M, Brotchie
Publikováno v:
Annals of neurology. 47(4 Suppl 1)
Treatment-related dyskinesias are the major limitation of dopamine replacement therapies such as levodopa in Parkinson's disease (PD). Recent studies in parkinsonian, nonhuman primates have highlighted abnormalities in neural functioning that might u
Autor:
M P, Hill, J M, Brotchie
Publikováno v:
British journal of pharmacology. 128(7)
1. The treatment of Parkinson's disease relies predominantly upon dopamine replacement therapy, usually with l-dihydroxyphenylalanine (L-DOPA). However, side-effects of long-term treatment, such as L-DOPA-induced dyskinesias can be more debilitating
Autor:
J M, Brotchie, S H, Fox
Publikováno v:
Movement disorders : official journal of the Movement Disorder Society. 14