Mitochondria-Associated Membranes (MAMs)

Autor: Patricia Gomez-Suaga, Sokhna M S Yakhine-Diop, Rosa A. González-Polo, Rubén Gómez-Sánchez, Mireia Niso-Santano, Mario Rodríguez-Arribas, José M. Fuentes, Guadalupe Martínez-Chacón, J. M Bravo San Pedro

Publikováno v: Rodríguez-Arribas, M, Yakhine-Diop, S M S, Pedro, J M B S, Gómez-Suaga, P, Gómez-Sánchez, R, Martínez-Chacón, G, Fuentes, J M, González-Polo, R A & Niso-Santano, M 2016, ' Mitochondria-Associated Membranes (MAMs) : Overview and Its Role in Parkinson’s Disease ', Molecular Neurobiology, pp. 1-17 . https://doi.org/10.1007/s12035-016-0140-8
Europe PubMed Central

Mitochondria-associated membranes (MAMs) are structures that regulate physiological functions between endoplasmic reticulum (ER) and mitochondria in order to maintain calcium signaling and mitochondrial biogenesis. Several proteins located in MAMs, i

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2d9954e0917bba269fe6c366106fb61
https://hdl.handle.net/11370/1b8197e2-8cb3-4d39-9ef2-7f33943e4722

Essential versus accessory aspects of cell death: recommendations of the NCCD 2015

Autor: Lorenzo Galluzzi, Thomas Rudel, Hans-Uwe Simon, Vishva M. Dixit, Erwin F. Wagner, Marie-Lise Gougeon, Andreas Linkermann, J M Bravo-San Pedro, Rosario Rizzuto, Cecília M. P. Rodrigues, Gian Maria Fimia, Hidenori Ichijo, Mathieu J.M. Bertrand, Kodi S. Ravichandran, Francis Ka-Ming Chan, Stephen W.G. Tait, Jochen H. M. Prehn, Richard A. Lockshin, Valina L. Dawson, Andreas Villunger, Sharad Kumar, Emily H. Cheng, Carlos López-Otín, Theocharis Panaretakis, Lucia Altucci, Gabriel A. Rabinovich, Michelangelo Campanella, Peter Vandenabeele, Marcus E. Peter, Francesco Cecconi, Noboru Mizushima, Ilio Vitale, Frank Madeo, Mikhail V. Blagosklonny, Zahra Zakeri, Stuart A. Aaronson, Gabriel Núñez, Eric H. Baehrecke, Nektarios Tavernarakis, Gyorgy Szabadkai, Eleonora Candi, Brent R. Stockwell, Dale E. Bredesen, Seamus J. Martin, Thomas Kaufmann, Sonia Melino, Dieter Adam, John M. Abrams, Katiuscia Bianchi, Yufang Shi, Emad S. Alnemri, Klas Blomgren, Pascal Meier, Catherine Brenner, Michael O. Hengartner, Philipp J. Jost, J M Hardwick, Eileen White, T Vanden Berghe, N. Di Daniele, Nicolas G. Bazan, H. L. Tang, Mauro Piacentini, V De Laurenzi, Beth Levine, Margherita Annicchiarico-Petruzzelli, Josef M. Penninger, Walter Malorni, Ted M. Dawson, Carmen Garrido, David W. Andrews, Douglas R. Green, György Hajnóczky, Jerry E. Chipuk, Wafik S. El-Deiry, Christoph Borner, Stuart A. Lipton, John A. Cidlowski, Klaus-Michael Debatin, Junying Yuan, Jan Paul Medema, Bertrand Joseph, Aaron Ciechanover, Ute M. Moll, Hinrich Gronemeyer, Paolo Pinton, Gerry Melino, Daniel J. Klionsky, Simone Fulda, John J. Lemasters, Cristina Muñoz-Pinedo, Hamsa Puthalakath, Navdeep S. Chandel, R De Maria, Jean-Christophe Marine, Richard A. Flavell, Brian David Dynlacht, W. G. Wood, Henning Walczak, David C. Rubinsztein, Guido Kroemer, Oliver Kepp, Richard A. Knight, Andrew Oberst, Enrico Lugli, J-C Martinou, Boris Zhivotovsky, Yoshihide Tsujimoto

Publikováno v: Cell death and differentiation
Cell Death and Differentiation
Kaufmann, Thomas; Simon, Hans-Uwe (2015). Essential versus accessory aspects of cell death: recommendations of the NCCD 2015. Cell death and differentiation, 22(1), pp. 58-73. Nature Publishing Group 10.1038/cdd.2014.137
CELL DEATH AND DIFFERENTIATION

Cells exposed to extreme physicochemical or mechanical stimuli die in an uncontrollable manner, as a result of their immediate structural breakdown. Such an unavoidable variant of cellular demise is generally referred to as ?accidental cell death' (A

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::86a0ef3dbb0451471d709e87ecf30256
http://www.nature.com/cdd/journal/v22/n1/full/cdd2014137a.html

Assessment of Glycolytic Flux and Mitochondrial Respiration in the Course of Autophagic Responses

Autor: V, Sica, J M, Bravo-San Pedro, F, Pietrocola, V, Izzo, M C, Maiuri, G, Kroemer, L, Galluzzi

Publikováno v: Methods in enzymology. 588

Autophagy is an evolutionarily conserved process that mediates prominent homeostatic functions, both at the cellular and organismal level. Indeed, baseline autophagy not only ensures the disposal of cytoplasmic entities that may become cytotoxic upon

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::89b5d6445d41792d02921e82d74dc990
https://pubmed.ncbi.nlm.nih.gov/28237099

Preface

Autor: L, Galluzzi, J M, Bravo-San Pedro, G, Kroemer

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::747c0690406ca527e03dacb5347efd56
https://doi.org/10.1016/s0076-6879(17)30075-7

Spermidine induces autophagy by inhibiting the acetyltransferase EP300

Autor: Mireia Niso-Santano, Maria Chiara Maiuri, V. Izzo, Guillermo Mariño, Guido Kroemer, David Enot, J M Bravo-San Pedro, Valentina Sica, Federico Pietrocola, Frank Madeo, S Lachkar

Several natural compounds found in health-related food items can inhibit acetyltransferases as they induce autophagy. Here we show that this applies to anacardic acid, curcumin, garcinol and spermidine, all of which reduce the acetylation level of cu

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::632ece483fa1f7f1b935b68645c95e2b
https://europepmc.org/articles/PMC4326581/