Zobrazeno 1 - 10
of 168
pro vyhledávání: '"J. J. Woodward"'
Publikováno v:
Journal of Neurochemistry. 72:1389-1393
NMDA receptors are potentiated by phosphorylation in a subunit- and kinase-specific manner. Both native and recombinant NMDA receptors are inhibited by behaviorally relevant concentrations of ethanol. Whether the phosphorylation state of individual s
Autor:
J J, WOODWARD
Publikováno v:
Bulletin. International Association of Medical Museums. 25
Autor:
J J, Woodward
Publikováno v:
Critical reviews in neurobiology. 14(1)
NMDA receptors are a multi-subunit family of ionotropic receptors activated by the neurotransmitter glutamate. Localized primarily postsynaptically in neurons, they play an important role in mediating excitatory synaptic neurotransmission and are imp
Publikováno v:
British journal of pharmacology. 131(7)
1. We have previously shown that toluene dose-dependently inhibits recombinant N-methyl-D-aspartate (NMDA) receptors at micromolar concentrations. This inhibition was rapid, almost complete and reversible. The NR1/2B combination was the most sensitiv
Autor:
J J, Woodward
Publikováno v:
Neurochemistry international. 35(2)
Autor:
J J, Woodward
Publikováno v:
Neurochemistry international. 35(2)
Publikováno v:
Alcoholism, clinical and experimental research. 23(2)
N-Methyl-D-aspartate (NMDA) receptors are ligand-gated ion channels that are important mediators of the actions of the excitatory amino acid neurotransmitter glutamate. Previous studies have shown that ethanol inhibits the function of both wild-type
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 286(1)
Previous studies have shown that toluene, which is commonly abused, depresses neuronal activity and causes behavioral effects in both animals and man similar to those observed for ethanol. In this study, the oocyte expression system was used to test
Autor:
J J, Woodward, S, Cueto
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 266(2)
N-Methyl-D-aspartic acid (NMDA, 500 microM) stimulated the net release of [3H]norepinephrine from rat hippocampal slices. The putative polyamine inverse agonist 1,10-diaminodecane (DA10) dose-dependently inhibited NMDA-stimulated release with a calcu
Publikováno v:
Alcohol and alcoholism (Oxford, Oxfordshire). Supplement. 1
N-methyl-D-aspartate (NMDA) stimulated a concentration dependent release of tritiated norepinephrine from cortical slices and tritiated and endogenous dopamine from striatal slices prepared from adult rat brain. Release of both neurotransmitters was