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of 7
pro vyhledávání: '"J. Ian Hertzler"'
Autor:
J Ian Hertzler, Jiajing Teng, Annabelle R Bernard, Michelle C Stone, Hannah L Kline, Gibarni Mahata, Nitish Kumar, Melissa M Rolls
Publikováno v:
PLoS Genetics, Vol 20, Iss 8, p e1011388 (2024)
Most neurons are not replaced after injury and thus possess robust intrinsic mechanisms for repair after damage. Axon injury triggers a calcium wave, and calcium and cAMP can augment axon regeneration. In comparison to axon regeneration, dendrite reg
Externí odkaz:
https://doaj.org/article/483122d715f74004b997f5e787ff4802
Publikováno v:
Developmental Biology. 497:18-25
Autor:
Derek M R Nye, Richard M Albertson, Alexis T Weiner, J Ian Hertzler, Matthew Shorey, Deborah C I Goberdhan, Clive Wilson, Kevin A Janes, Melissa M Rolls
Publikováno v:
PLoS Biology, Vol 18, Iss 3, p e3000657 (2020)
While many regulators of axon regeneration have been identified, very little is known about mechanisms that allow dendrites to regenerate after injury. Using a Drosophila model of dendrite regeneration, we performed a candidate screen of receptor tyr
Externí odkaz:
https://doaj.org/article/f335bd2fb73341978ccd9bb1deec833d
Autor:
Deborah C.I. Goberdhan, Derek Nye, J. Ian Hertzler, Richard M. Albertson, Kevin A. Janes, Alexis T. Weiner, Clive Wilson, Matthew Shorey, Melissa M. Rolls
Publikováno v:
PLoS Biology, Vol 18, Iss 3, p e3000657 (2020)
PLoS Biology
PLoS Biology
While many regulators of axon regeneration have been identified, very little is known about mechanisms that allow dendrites to regenerate after injury. Using a Drosophila model of dendrite regeneration, we performed a candidate screen of receptor tyr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e9ab93d6841c1cb8742e3325a77d757
https://ora.ox.ac.uk/objects/uuid:939e2050-3287-4c71-be79-f84ec9f635e0
https://ora.ox.ac.uk/objects/uuid:939e2050-3287-4c71-be79-f84ec9f635e0
Autor:
Amber R. Smith, J. Ian Hertzler, Molly O. Novak, Zackery J. Barile, Sidney L. Satterfield, Michael J. Palladino, Alejandra C. Gutierrez, Keri J. Fogle, Joy H. Shon, Caleb S. McCardell
Seizures are a feature not only of the many forms of epilepsy, but also of global metabolic diseases such as mitochondrial encephalomyopathy (ME) and glycolytic enzymopathy (GE). Modern anti-epileptic drugs (AEDs) are successful in many cases, but so
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36242048bf137aafe790e8d2ca4915db
https://europepmc.org/articles/PMC6536302/
https://europepmc.org/articles/PMC6536302/
Publikováno v:
Journal of Neurogenetics. 30:247-258
Effective therapies are lacking for mitochondrial encephalomyopathies (MEs). MEs are devastating diseases that predominantly affect the energy-demanding tissues of the nervous system and muscle, causing symptoms such as seizures, cardiomyopathy, and
Akademický článek
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