Zobrazeno 1 - 5
of 5
pro vyhledávání: '"J. Han M. Levels"'
Autor:
Lars E. Larsen, Marjolein A.W. van den Boogert, Wilson A. Rios-Ocampo, Jos C. Jansen, Donna Conlon, Patrick L.E. Chong, J. Han M. Levels, Roos E. Eilers, Vinay V. Sachdev, Noam Zelcer, Tobias Raabe, Miao He, Nicholas J. Hand, Joost P.H. Drenth, David J. Rader, Eric S.G. Stroes, Dirk J. Lefeber, Johan W. Jonker, Adriaan G. Holleboom
Publikováno v:
Cellular and Molecular Gastroenterology and Hepatology, Vol 13, Iss 2, Pp 583-597 (2022)
Background & Aims: Recently, novel inborn errors of metabolism were identified because of mutations in V-ATPase assembly factors TMEM199 and CCDC115. Patients are characterized by generalized protein glycosylation defects, hypercholesterolemia, and f
Externí odkaz:
https://doaj.org/article/f83a6ee89be64fb6964bdd0493f8352e
Autor:
David J. Rader, Joost P.H. Drenth, Adriaan G. Holleboom, Jos C. Jansen, J. Han M. Levels, Lars E. Larsen, Marjolein A.W. van den Boogert, Roos E. Eilers, Patrick L.E. Chong, Donna M. Conlon, Vinay Sachdev, Nicholas J. Hand, Wilson A. Rios-Ocampo, Eric S.G. Stroes, Miao He, Noam Zelcer, Tobias Raabe, Dirk Lefeber, Johan W. Jonker
Publikováno v:
Cellular and molecular gastroenterology and hepatology, 13(2), 583-597. Elsevier Inc.
Cellular and Molecular Gastroenterology and Hepatology
Cellular and Molecular Gastroenterology and Hepatology, Vol 13, Iss 2, Pp 583-597 (2022)
Cellular and Molecular Gastroenterology and Hepatology, 13, 583-597
Cellular and molecular gastroenterology and hepatology, 13(2), 583-597. HANLEY & BELFUS-ELSEVIER INC
Cellular and Molecular Gastroenterology and Hepatology, 13, 2, pp. 583-597
Cellular and Molecular Gastroenterology and Hepatology
Cellular and Molecular Gastroenterology and Hepatology, Vol 13, Iss 2, Pp 583-597 (2022)
Cellular and Molecular Gastroenterology and Hepatology, 13, 583-597
Cellular and molecular gastroenterology and hepatology, 13(2), 583-597. HANLEY & BELFUS-ELSEVIER INC
Cellular and Molecular Gastroenterology and Hepatology, 13, 2, pp. 583-597
Background & Aims Recently, novel inborn errors of metabolism were identified because of mutations in V-ATPase assembly factors TMEM199 and CCDC115. Patients are characterized by generalized protein glycosylation defects, hypercholesterolemia, and fa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd690ee2f3528274a974978110078931
https://pure.amc.nl/en/publications/defective-lipid-dropletlysosome-interaction-causes-fatty-liver-disease-as-evidenced-by-human-mutations-in-tmem199-and-ccdc115(4a8f6f40-8c9d-48bf-9fc0-f57814f0e83b).html
https://pure.amc.nl/en/publications/defective-lipid-dropletlysosome-interaction-causes-fatty-liver-disease-as-evidenced-by-human-mutations-in-tmem199-and-ccdc115(4a8f6f40-8c9d-48bf-9fc0-f57814f0e83b).html
Autor:
Erin M. Foley, Michiel A. J. van de Sande, Julia J. Witjes, Michael W.T. Tanck, J. Han M. Levels, Philip L.S.M. Gordts, Erik S.G. Stroes, Max Nieuwdorp, Marjolein A.W. van den Boogert, Geesje M. Dallinga-Thie, Jeff Esko, Kristin I. Stanford, H. Carlijne Hassing, Hans L. Mooij, Sophie J. Bernelot Moens, John J.P. Kastelein
Publikováno v:
Mooij, H L, Moens, S J, Gordts, P L, Stanford, K I, Foley, E M, van den Boogert, M A, Witjes, J J, Hassing, H, Tanck, M W, van de Sande, M A, Levels, J, Kastelein, J J, Stroes, E S, Dallinga-Thie, G M, Esko, J D & Nieuwdorp, M 2015, ' Ext1 heterozygosity causes a modest effect on postprandial lipid clearance in humans ', Journal of Lipid Research, vol. 56, no. 3, pp. 665-673 . https://doi.org/10.1194/jlr.M053504
Journal of Lipid Research, 56(3), 665-673. American Society for Biochemistry and Molecular Biology Inc.
Journal of lipid research, vol 56, iss 3
Journal of Lipid Research, Vol 56, Iss 3, Pp 665-673 (2015)
Journal of Lipid Research, 56(3), 665-673
Journal of lipid research, 56(3), 665-673. American Society for Biochemistry and Molecular Biology Inc.
Journal of Lipid Research, 56(3), 665-673. American Society for Biochemistry and Molecular Biology Inc.
Journal of lipid research, vol 56, iss 3
Journal of Lipid Research, Vol 56, Iss 3, Pp 665-673 (2015)
Journal of Lipid Research, 56(3), 665-673
Journal of lipid research, 56(3), 665-673. American Society for Biochemistry and Molecular Biology Inc.
Elevated nonfasting TG-rich lipoprotein levels are a risk factor for CVD. To further evaluate the relevance of LDL-receptor (LDLr) pathway and heparan sulfate proteoglycans (HSPGs) in TG homeostasis, we analyzed fasting and postprandial TG levels in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5af6964f87d47fc4768490da71ab486
https://research.vumc.nl/en/publications/b999cf6c-e2a1-4deb-a341-20c9f4c27e26
https://research.vumc.nl/en/publications/b999cf6c-e2a1-4deb-a341-20c9f4c27e26
Publikováno v:
Proteomics, 6(2), 721-730. Wiley-VCH Verlag
Plasma lipoproteins, such as high-density lipoprotein (HDL), can serve as carriers for a wide range of proteins that are involved in processes such as lipid metabolism, thrombosis, inflammation and atherosclerosis. The identification of HDL-associate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b6fe1f060a328f799a7c7210285482f1
https://doi.org/10.1002/pmic.200500191
https://doi.org/10.1002/pmic.200500191
Publikováno v:
Proteomics; Jan2006, Vol. 6 Issue 2, p721-730, 10p