Zobrazeno 1 - 10
of 13
pro vyhledávání: '"J. G. Tidball"'
Autor:
H. X Nguyen, J. G Tidball
Current evidence indicates that the physiological functions of inflammatory cells are highly sensitive to their microenvironment, which is partially determined by the inflammatory cells and their potential targets. In the present investigation, inter
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f632b1ab952773c1564f5967a8009bf7
https://europepmc.org/articles/PMC2342622/
https://europepmc.org/articles/PMC2342622/
Autor:
J G, Tidball, E, Berchenko
Publikováno v:
Journal of leukocyte biology. 65(4)
Previous observations have shown that neutrophil invasion precedes macrophage invasion during muscle inflammation and that peak muscle injury is observed at the peak of ED1+ macrophage invasion. We tested the hypothesis that neutrophil invasion cause
Publikováno v:
Journal of leukocyte biology. 64(4)
The objective of this study was to determine the role of nitric oxide in muscle inflammation, fiber necrosis, and apoptosis of inflammatory cells in vivo. The effects of nitric oxide synthase (NOS) inhibition on the concentrations of neutrophils, ED1
Publikováno v:
Journal of cellular biochemistry. 64(1)
In the present investigation, we have tested the hypothesis that calpain expression or activity in skeletal muscle is influenced by changes in mechanical loading in vivo. Muscle unloading for 10 days produced no change in the concentrations of calpai
Publikováno v:
Mineral and electrolyte metabolism. 21(1-3)
Parathyroid hormone (PTH)-induced osteoblast retraction may play a pivotal role a role in bone resorption by providing osteoclasts direct access to mineralized bone surface. We have been working on the hypothesis that the calpains participate in this
Autor:
D J, Law, J G, Tidball
Publikováno v:
The American journal of pathology. 142(5)
The myotendinous junction (MTJ) is the major site of force transmission from myofibrils across the muscle cell membrane to the extracellular matrix. The MTJ is thus an appropriate model system in which to test the hypothesis that dystrophin, the gene
Publikováno v:
Biochemistry and molecular biology international. 29(5)
Calcium-activated neutral protease activity was detected in mouse MC3T3-E1 cell extracts. Inclusion of the cysteine protease inhibitor, E64c, reduced the activity, while pretreatment of intact cells with 10 nM parathyroid hormone for 90 minutes incre
Autor:
J G, Tidball
Publikováno v:
Exercise and sport sciences reviews. 19
Autor:
J G, Tidball, D J, Law
Publikováno v:
The American journal of pathology. 138(1)
Dystrophin, the deficient gene product in Duchenne muscular dystrophy, is located subjacent to the muscle cell membrane at myotendinous junctions, as well as along the entire muscle cell. Myotendinous junctions are sites at which thin filaments norma
Autor:
E. V. Cheung, J. G. Tidball
Publikováno v:
Inflammation Research; Apr2003, Vol. 52 Issue 4, p170-176, 7p