Zobrazeno 1 - 10
of 36
pro vyhledávání: '"J. D Taurog"'
Autor:
W A Simmons, S G Summerfield, D C Roopenian, C A Slaughter, A R Zuberi, S J Gaskell, R S Bordoli, J Hoyes, C R Moomaw, R A Colbert, L Y Leong, G W Butcher, R E Hammer, J D Taurog
Publikováno v:
The Journal of Immunology. 159:2750-2759
We have identified two peptides corresponding to the male-specific HY minor histocompatibility Ags presented by HLA-B27 in transgenic rodents, isolated from whole cell extracts and from immunoprecipitated B27 molecules of male B27 rat spleen cells. H
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0709cb99394a0327c8ddec28092c5ab9
https://doi.org/10.4049/jimmunol.159.6.2750
https://doi.org/10.4049/jimmunol.159.6.2750
Autor:
M Breban, J L Fernández-Sueiro, J A Richardson, R R Hadavand, S D Maika, R E Hammer, J D Taurog
Publikováno v:
The Journal of Immunology. 156:794-803
Rats transgenic for the human MHC class I gene HLA-B27 are susceptible to a spontaneous multisystem inflammatory disease that resembles human B27-associated disease. This disease requires a high level of expression of the B27 transgene product in cel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c77b57834a719e0c7c142d791ed16481
https://doi.org/10.4049/jimmunol.156.2.794
https://doi.org/10.4049/jimmunol.156.2.794
Publikováno v:
Clinical and experimental rheumatology. 27(2)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::dfcdf71c3d8db1caede93ddf6c6d1b92
https://pubmed.ncbi.nlm.nih.gov/19473590
https://pubmed.ncbi.nlm.nih.gov/19473590
Publikováno v:
The Journal of Immunology. 144:1512-1517
The class I MHC molecule HLA-B27 bears an unpaired Cys residue at position 67, which is predicted to face the Ag binding pocket, based on the x-ray crystallographic model of HLA-A2. To investigate the potential of this residue in the antigenic struct
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::89fb83036c1672640798a3692407751a
https://doi.org/10.4049/jimmunol.144.4.1512
https://doi.org/10.4049/jimmunol.144.4.1512
Autor:
H E, Krug, J D, Taurog
Publikováno v:
The Journal of rheumatology. 27(5)
To investigate whether HLA-B27 influences the expression of murine progressive ankylosis (MPA), a single-gene autosomal recessive mouse model of ankylosing spondylitis that arises in mice homozygous for the ank gene.Mice transgenic for HLA-B27 were b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::de014ba1432390ae1596caef79686594
https://pubmed.ncbi.nlm.nih.gov/10813297
https://pubmed.ncbi.nlm.nih.gov/10813297
Autor:
S, Baggia, J L, Lyons, E, Angell, A, Barkhuizen, Y B, Han, S R, Planck, J D, Taurog, J T, Rosenbaum
Publikováno v:
Journal of investigative medicine : the official publication of the American Federation for Clinical Research. 45(5)
Humans with the major histocompatibility antigen B27 (HLA-B27) are especially at risk for developing rheumatic disorders such as ankylosing spondylitis and Reiter's syndrome. Acute anterior uveitis (AAU) often occurs in association with these disease
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::594d995b7a70f15e6e3452a58bb3e4a7
https://pubmed.ncbi.nlm.nih.gov/9250003
https://pubmed.ncbi.nlm.nih.gov/9250003
Publikováno v:
The Journal of rheumatology. 24(1)
Murine progressive ankylosis is an autosomal recessive disorder in mice similar to the human spondyloarthropathies. The gene responsible for progressive ankylosis, ank, has not been identified and its product is unknown. We investigated whether the i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ff18fd42a18b214f05431a9c909df4dd
https://pubmed.ncbi.nlm.nih.gov/9002021
https://pubmed.ncbi.nlm.nih.gov/9002021
Publikováno v:
The American journal of pathology. 149(5)
Germfree rats transgenic for the human genes HLA-B27 and beta 2-microglobulin were colonized with hemolysin-positive (Hly+) or hemolysin-negative (Hly-) strains of Listeria monocytogenes. HLA-B27 rats were very susceptible to infection with Hly+ L mo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b271829341b8c5424a4a43e222ec59ca
https://pubmed.ncbi.nlm.nih.gov/8909262
https://pubmed.ncbi.nlm.nih.gov/8909262
Autor:
J D Taurog, Kenneth H. Wilson, T E Hamm, Heiko C. Rath, Robert E. Hammer, J S Ikeda, Hans H Herfarth, W B Grenther, Edward Balish, Ryan Balfour Sartor
Genetic and environmental factors are important in the pathogenesis of clinical and experimental chronic intestinal inflammation. We investigated the influence of normal luminal bacteria and several groups of selected bacterial strains on spontaneous
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d1df5f9dbbd219fe60bcc6eb235c24ad
https://europepmc.org/articles/PMC507509/
https://europepmc.org/articles/PMC507509/
Autor:
M, Breban, J L, Fernández-Sueiro, J A, Richardson, R R, Hadavand, S D, Maika, R E, Hammer, J D, Taurog
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 156(2)
Rats transgenic for the human MHC class I gene HLA-B27 are susceptible to a spontaneous multisystem inflammatory disease that resembles human B27-associated disease. This disease requires a high level of expression of the B27 transgene product in cel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::466f3486ef314c91788c42e7fb537e32
https://pubmed.ncbi.nlm.nih.gov/8543835
https://pubmed.ncbi.nlm.nih.gov/8543835