Zobrazeno 1 - 10
of 128
pro vyhledávání: '"J. Stechschulte"'
Autor:
Joshua T. Lackey, MD, Erin P. Murray, MD, Mark J. Winston, MD, Daniel J. Stechschulte, Jr., MD, PhD
Publikováno v:
Journal of Hand Surgery Global Online, Vol 4, Iss 5, Pp 295-298 (2022)
A 16-year-old boy sustained a complete, closed, intrasubstance rupture of both heads of the biceps brachii after a rope swing accident. The patient was managed with open direct repair of the muscle belly. After the surgery, he underwent an extensive
Externí odkaz:
https://doaj.org/article/007976330dcd4fccba96489655fadc00
Publikováno v:
Mediators of Inflammation, Vol 13, Iss 5-6, Pp 335-341 (2004)
STUDIES were carried out to evaluate the impact of a high-fat dietary regimen on aortic wall thickness, peripheral blood leukocyte profile, and plasma cholesterol and triglyceride levels in the mast cell-deficient Sl/Sld mouse. The results demonstrat
Externí odkaz:
https://doaj.org/article/3e88824820334023b4487c7791720aca
Publikováno v:
Mediators of Inflammation, Vol 12, Iss 3, Pp 147-155 (2003)
Coronary heart disease secondary to atherosclerosis is still the leading cause of death in the US. Animal models used for elucidating the pathogenesis of this disease primarily involve rabbits and pigs. Previous studies from this laboratory have demo
Externí odkaz:
https://doaj.org/article/33b3c52b7e0c45c49caa5aeea5a7db40
Publikováno v:
Mediators of Inflammation, Vol 12, Iss 2, Pp 79-87 (2003)
Background: We have previously shown that incubation of human endothelial cells with mast cell granules results in potentiation of lipopolysaccharide-induced production of interleukin-6 and interleukin-8.
Externí odkaz:
https://doaj.org/article/73f6057d76ef470c9910473741b33f8a
Publikováno v:
Mediators of Inflammation, Vol 7, Iss 5, Pp 355-361 (1998)
Previous studies have shown that mast cell granules (MCG) inhibit numerous macrophage functions including tumour cytotoxicity, superoxide and nitric oxide (NO) production, and FCγ2a receptor-mediated phagocytosis. In this study, the effect of MCG on
Externí odkaz:
https://doaj.org/article/f68276d1d3c24c7ba24e6956c0686864
Publikováno v:
Mediators of Inflammation, Vol 7, Iss 6, Pp 397-407 (1998)
The exposure of the macrophage cell line, J774 to mast cell granules (MCG) led to the form ation of altered nuclear transcription factor proteins (NFκBx), which had faster electrophoretic mobility than the p50 homodimer of NF-κB, but retained compa
Externí odkaz:
https://doaj.org/article/27b95133ecf14057976387fe0f1627f5
Publikováno v:
Mediators of Inflammation, Vol 4, Iss 6, Pp 406-412 (1995)
The mechanism by which phagocytosed mast cell granules (MCGs) inhibit macrophage superoxide production has not been defined. In this study, rat peritoneal macrophages were co-incubated with either isolated intact MCGs or MCG-sonicate, and their respi
Externí odkaz:
https://doaj.org/article/1f12f38a6dfd4652982325292cad4e57
Autor:
Damandeep S. Walia, Mukut Sharma, Vineesh V. Raveendran, Jianping Zhou, Ram Sharma, Daniel J. Stechschulte, Kottarappat N. Dileepan
Publikováno v:
Mediators of Inflammation, Vol 2012 (2012)
We examined the effect of intact human mast cells (HMC-1 5C6) and their selected mediators on interleukin-6 (IL-6) production and bone morphogenetic protein-2 (BMP-2) expression in human coronary artery endothelial cells (HCAEC) in the presence and a
Externí odkaz:
https://doaj.org/article/cb0ceae9e5994d84a5245abfb18ce4c6
Autor:
Timothy J. Nickel, Mohammad H. Kabir, Jaya Talreja, Daniel J. Stechschulte, Kottarappat N. Dileepan
Publikováno v:
Mediators of Inflammation, Vol 2006 (2006)
Protease-activated receptors (PARs) are G-protein-coupled receptors which initiate inflammatory responses when activated by specific serine proteases. This study was conducted to examine whether human conjunctival epithelial cells (HCECs) express fun
Externí odkaz:
https://doaj.org/article/119ef09cbfc248c9bc1f123243dde83f
Autor:
Xiaoyu Tan, Beth Levant, Joyce G. Slusser, Matthew E. Sweeney, Vineesh V. Raveendran, Kottarappat N. Dileepan, Daniel J. Stechschulte
Publikováno v:
Immunology. 132:578-588
Summary Histamine is a well-recognized modulator of vascular inflammation. We have shown that histamine, acting via H1 receptors (H1R), synergizes lipopolysaccharide (LPS)-induced production of prostaglandin I(2) (PGI(2)), PGE(2) and interleukin-6 (I