Zobrazeno 1 - 10
of 74
pro vyhledávání: '"J-P Rousset"'
Autor:
T. Mazingue, M. Lomello-Tafin, M. Passard, C. Hernandez-Rodriguez, L. Goujon, J.-L. Rousset, F. Morfin, J.-F. Laithier
Publikováno v:
Journal of Sensors and Sensor Systems, Vol 3, Iss 2, Pp 273-280 (2014)
Palladium platinum (PdPt) has been intensively studied these last decades due to high conversion rate in hydrogen oxidation at room temperature with significant exothermic effects. These remarkable properties have been studied by measuring the temper
Externí odkaz:
https://doaj.org/article/7fc0ea4cfe1d4e309a191e56cba57877
Autor:
I. Sermet-Gaudelus, M. Renouil, A. Fajac, L. Bidou, B. Parbaille, S. Pierrot, N. Davy, E. Bismuth, P. Reinert, G. Lenoir, J. F. Lesure, J. P. Rousset, A. Edelman
Publikováno v:
BMC Medicine, Vol 16, Iss 1, Pp 1-1 (2018)
The original article [1] contains errors in Table 1 affecting some of the presented oligonucleotide sequences and readthrough values in Table 1.
Externí odkaz:
https://doaj.org/article/f79311b8b37c4cfd84d44c568f60e93a
Autor:
Jean François Lesure, A. Fajac, N. Davy, Isabelle Sermet-Gaudelus, L. Bidou, Michel Renouil, S. Pierrot, Gérard Lenoir, J. P. Rousset, B. Parbaille, Elise Bismuth, Aleksander Edelman, P. Reinert
Publikováno v:
BMC Medicine, Vol 16, Iss 1, Pp 1-1 (2018)
BMC Medicine
BMC Medicine
Background Cystic fibrosis (CF) is caused by mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR) protein, which acts as a chloride channel activated by cyclic AMP (cAMP). The most frequent mutation found in 7
Publikováno v:
Gene Therapy
Gene Therapy, Nature Publishing Group, 2004, 11, pp.619-627
Gene Therapy, Nature Publishing Group, 2004, 11, pp.619-627
The suppression levels induced by gentamicin on premature stop codons, caused by primary nonsense mutations found in muscular dystrophy patients, were assessed using a very sensitive dual reporter gene assay. Results show that: (i) the effect of gent
Publikováno v:
Molekuliarnaia biologiia. 41(6)
In universal-code eukaryotes, a single class-1 translation termination factor eRF1 decodes all three stop codons, UAA, UAG, and UGA. In some ciliates with variant genetic codes one or two stop codons are used to encode amino acid(s) and are not recog
Publikováno v:
Molecular and General Genetics MGG. 222:291-296
We present a strategy to elucidate the rate-limiting steps in activation of carcinogenic compounds by cytochromes P450. The principle was to select Reuber rat hepatoma cells for resistance to a procarcinogen. The hypothesis was that resistant cells s
Publikováno v:
Cellular and molecular biology (Noisy-le-Grand, France).
A prion is an infectious, altered form of a cellular protein which can self-propagate and affect normal phenotype. Prion conversion has been observed for mammalian and yeast proteins but molecular mechanisms that trigger this process remain unclear.
Autor:
M, Cassan, J P, Rousset
Publikováno v:
BMC Molecular Biology
Background Translation termination is mediated through an interaction between the release factors eRF1 and eRF3 and the stop codon within its nucleotide context. Although it is well known that the nucleotide contexts both upstream and downstream of t
A frameshift event is necessary for expression of the products of the pol gene in a number of retroviruses, including human immunodeficiency virus type 1 (HIV-1). The basic signals necessary for frameshifting consist of a shifty sequence in which the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::845840edecdfaeb9118773a97e2618f6
https://europepmc.org/articles/PMC236606/
https://europepmc.org/articles/PMC236606/