Zobrazeno 1 - 4
of 4
pro vyhledávání: '"J W Homeister"'
Publikováno v:
The Journal of Immunology. 150:1055-1064
The purpose of this study was to determine if recombinant human soluble CR1 (sCR1) could prevent tissue damage associated with the activation of human complement. Directly mediated human complement-dependent myocardial injury was induced in the rabbi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8cf5ace5715aac1a21975dca602c3693
https://doi.org/10.4049/jimmunol.150.3.1055
https://doi.org/10.4049/jimmunol.150.3.1055
Publikováno v:
Blood. 92(7)
Selectin adhesion molecules mediate leukocyte rolling on activated endothelium, a prerequisite to leukocyte accumulation at sites of inflammation. The precise role of each selectin (E-, P-, and L-) in this process is unclear and may vary depending on
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::650706c231bf9727ac566fa0ff39dd10
https://pubmed.ncbi.nlm.nih.gov/9746773
https://pubmed.ncbi.nlm.nih.gov/9746773
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 150(3)
The purpose of this study was to determine if recombinant human soluble CR1 (sCR1) could prevent tissue damage associated with the activation of human complement. Directly mediated human complement-dependent myocardial injury was induced in the rabbi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2b65b49a6ceba0b6a5749817fa1fef25
https://pubmed.ncbi.nlm.nih.gov/8423331
https://pubmed.ncbi.nlm.nih.gov/8423331
Publikováno v:
Federation proceedings. 46(1)
Considerable attention is being given to the interactions that occur among blood platelets, neutrophils, and the vascular endothelium. There is an increasing awareness that the various blood elements interact in the process of thrombus formation and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8a767678bc50eb44221d79bdf0639489
https://pubmed.ncbi.nlm.nih.gov/3542578
https://pubmed.ncbi.nlm.nih.gov/3542578