Zobrazeno 1 - 7
of 7
pro vyhledávání: '"J T, Sullebarger"'
Autor:
Mark N. Allen, Gerson S. Lichtenberg, Shimon A. Reisner, P. G. Fitzpatrick, Richard S. Meltzer, J. T. Sullebarger, Ling S. Ong
Publikováno v:
European Heart Journal. 13:389-394
To evaluate the applicability of myocardial contrast echocardiography for the assessment of coronary blood flow reserve, 21 consecutive patients undergoing coronary angiography were studied. Only patients with a single left anterior descending lesion
Publikováno v:
Journal of Applied Physiology. 69:962-967
Phenylephrine (PE) bolus and infusion methods have both been used to measure baroreflex sensitivity in humans. To determine whether the two methods produce the same values of baroreceptor sensitivity, we administered intravenous PE by both bolus inje
Autor:
Chi-Ling Hang, J. T. Sullebarger
Publikováno v:
Catheterization and Cardiovascular Diagnosis. 28:228-230
This report describes an unusual case of patent ductus arteriosus, presenting in old age. The patient is the oldest living female with patent ductus reported thus far in the medical literature [1,2]. She initially presented with chest pressure and dy
Publikováno v:
Catheterization and cardiovascular interventions : official journal of the Society for Cardiac AngiographyInterventions. 46(1)
Degenerative disease of aortocoronary saphenous vein grafts is a major cause of late morbidity and mortality in patients after coronary bypass surgery. We previously described a technique for recanalization of totally occluded grafts using extraction
Publikováno v:
Cardiovascular research. 32(5)
The present investigation was designed to determine if atrial natriuretic peptide (ANP) gene expression increases in extracardiac as well as within the heart in congestive heart failure.Congestive heart failure (CHF) was induced by producing cardiac
Autor:
L. Guthinger, J. T. Sullebarger, R. P. Frantz, S. Sakamoto, Chang-seng Liang, Tai-Hwang M. Fan, M. Suematsu
Publikováno v:
Circulation. 84(1)
BACKGROUND The reductions of myocardial beta-adrenergic receptor density and responsiveness to catecholamines in congestive heart failure are associated with excessive sympathetic stimulation. The purpose of this study was to determine whether the my
Publikováno v:
Journal of Clinical Investigation. 84:1267-1275
The reduction of myocardial beta-adrenoceptor density in congestive heart failure has been thought to be caused by agonist-induced homologous desensitization. However, recent evidence suggests that excessive adrenergic stimulation may not produce myo