Zobrazeno 1 - 10
of 404
pro vyhledávání: '"J T, Hoff"'
Autor:
G. M. Teasdale
Publikováno v:
British Journal of Surgery. 73:328-328
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::878384be46f9b1a6b4f12d881d61a909
https://doi.org/10.1002/bjs.1800730437
https://doi.org/10.1002/bjs.1800730437
Autor:
Teasdale, G M
Publikováno v:
British Journal of Surgery; April 1986, Vol. 73 Issue: 4 p328-328, 1p
Publikováno v:
British Journal of Neurosurgery. 21:224-227
We present a case showing the development of a Schmorl's node from its acute to chronic stage by serial MRI. In contrast to the mature Schmorl's node, imaging characteristics of the acute Schmorl's node have not been well-illustrated, particularly by
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10d37812e9542c6721a6f960d4099c62
https://doi.org/10.1080/02688690701317169
https://doi.org/10.1080/02688690701317169
Publikováno v:
Acta neurochirurgica. Supplement. 102
New protein synthesis is key to ischemic tolerance induced by preconditioning and ribosomal protein S6 kinases (p70 S6 K) are important enzymes in protein synthesis. Hyperbaric oxygen preconditioning (HBOP) reduces ischemic brain damage. This study i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::43a64bedabdcac22063f36202fa8737a
https://pubmed.ncbi.nlm.nih.gov/19388337
https://pubmed.ncbi.nlm.nih.gov/19388337
Publikováno v:
Acta neurochirurgica. Supplement. 105
Intracerebral hemorrhage (ICH) is a subtype of stroke with very high mortality. Experiments have indicated that clot lysis and iron play an important role in ICH-induced brain injury. Iron overload occurs in the brain after ICH in rats. Intracerebral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7b64b3a932373d1d0af762a9e6802c6a
https://pubmed.ncbi.nlm.nih.gov/19066072
https://pubmed.ncbi.nlm.nih.gov/19066072
Publikováno v:
Acta neurochirurgica. Supplement. 105
In this paper, we review current knowledge on blood-brain barrier (BBB) dysfunction following intracerebral hemorrhage (ICH). BBB disruption is a hallmark of ICH-induced brain injury. Such disruption contributes to edema formation, the influx of leuk
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d0b52c000caa4a9299b3787aa7b1ddd8
https://pubmed.ncbi.nlm.nih.gov/19066086
https://pubmed.ncbi.nlm.nih.gov/19066086
Publikováno v:
Acta neurochirurgica. Supplement. 96
Hemoglobin degradation products result in brain injury after intracerebral hemorrhage (ICH). Recent studies found that intracerebral infusion of heme oxygenase inhibitors reduces hemoglobin- and ICH-induced brain edema in rats and pigs. The present s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c9fed8a1113b2fd55fcc4753ff76088a
https://pubmed.ncbi.nlm.nih.gov/16671461
https://pubmed.ncbi.nlm.nih.gov/16671461
Publikováno v:
Acta neurochirurgica. Supplement. 96
Iron overload occurs in brain after intracerebral hemorrhage (ICH). Deferoxamine, an iron chelator, attenuates perihematomal edema and oxidative stress in brain after ICH. We investigated the effects of deferoxamine on cerebrospinal fluid (CSF) free
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2c73cd527bf1a79a3f5bf2e2a42c8ac8
https://pubmed.ncbi.nlm.nih.gov/16671454
https://pubmed.ncbi.nlm.nih.gov/16671454
Autor:
Sonia V. Eden, Devin L. Brown, Lewis B. Morgenstern, Darin B. Zahuranec, Melinda A. Smith, Nicole R. Gonzales, Paxton J. Longwell, J. T. Hoff, Lynda D. Lisabeth, Nelda M. Garcia
Publikováno v:
Journal of neurology, neurosurgery, and psychiatry. 77(3)
Background: Studies on intracerebral haemorrhage (ICH) from tertiary care centres may not be an accurate representation of the true spectrum of disease presentation. Objective: To describe the clinical and imaging presentation of ICH in a community d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0bb2b678f3e1a35f60161d5fd0ceac6a
https://pubmed.ncbi.nlm.nih.gov/16484640
https://pubmed.ncbi.nlm.nih.gov/16484640