Zobrazeno 1 - 10
of 12
pro vyhledávání: '"J T, Berg"'
Autor:
Scott P. Orr, J. T. Berg, Paula L. Castro-Chapman, Suzanne L. Pineles, Kristen Salomon, Jessica Yanson
Publikováno v:
Psychiatry Research. 261:574-580
For over three decades, there has been considerable research documenting increased physiological reactivity to trauma-related stimuli as a characteristic feature of posttraumatic stress disorder (PTSD). The present study explored the potential for ph
Autor:
J. T. Berg
Publikováno v:
Inflammation Research. 55:565-571
To determine whether zymosan priming protects rats against oxygen toxicity. 37 rats were used with 6 treatment groups. 11 received zymosan priming (3 daily i. v. injections with 15 mg zymosan and 2 days rest) before 52 h exposure to either normobaric
Publikováno v:
Journal of Endotoxin Research. 1:108-113
Previous studies have shown that diphosphoryl lipid A (DPL), but not monophosphoryl lipid A (MPL), protects rats against lethal hyperoxia. However, the mechanism of DPL-induced 02 resistance is not clear. In this study, we demonstrated that tracheal
Publikováno v:
Journal of Applied Physiology. 74:2812-2819
Alveolar macrophages (AM) play an important role in lung biology. In this study, we demonstrated that tracheal insufflation of liposome-encapsulated dichloromethylene diphosphonate (Cl2MDP-liposome) selectively depleted AMs in rats. Insufflation of a
Publikováno v:
Experimental Biology and Medicine. 193:167-170
Research on endotoxin protection from oxygen toxicity is presently limited to the rat model since only rats have been protected by endotoxin. This study reports that endotoxin also extends survival of adult male mice in hyperoxia (greater than 99% ox
Publikováno v:
Journal of Applied Physiology. 68:549-553
Treatment with endotoxin protects rats against lung injury during hyperoxia (greater than 98% oxygen at 1 atmosphere absolute for 60 h). This study demonstrates that serum from endotoxin-treated donor rats also protects recipients from oxygen toxicit
Publikováno v:
Pharmacotherapy. 16(6)
Warfarin is a commonly used oral anticoagulant that is usually initiated after the definitive diagnosis of a certain thromboembolic disorder or disease. Warfarin therapy will usually be prescribed for 6-12 weeks or more, and some patients may continu
Publikováno v:
Experimental lung research. 21(1)
Recently an alveolar macrophage (AM)-depleted rat model has been characterized and it has been demonstrated that AM are required for the endotoxin-induced tumor necrosis factor (TNF) release into the alveolar space (J Appl Physiol 1993;74:2812-2819).
Publikováno v:
The American journal of physiology. 266(1 Pt 1)
Endotoxin and the cytokines, tumor necrosis factor and interleukin-1, are known to protect adult rats against O2 toxicity. However, whether the effect of endotoxin is mediated through its direct effect on lung cells or through cytokines is not clear.
Autor:
J T, Berg, R M, Smith
Publikováno v:
Research communications in chemical pathology and pharmacology. 47(2)
Endotoxin (1 mg/kg, I.P.) greatly reduces lung damage and pleural edema in rats exposed to greater than 99% oxygen. Endotoxin also activates and depletes complement in vitro. Prior in vivo complement depletion has been shown to protect rats from lung