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Autor:
J N Yang, Y T Tratsiakovich, T J Jiao, A M Mahdi, J T Tengbom, Z Z Zhou, A C Collado, J L Lundberg, J P Pernow
Publikováno v:
European Heart Journal. 43
Background Arginase is involved in the development of ischemia-reperfusion injury by regulating nitric oxide (NO) bioactivity via competition with NO synthase (NOS) for their common substrate L-arginine. Erythrocytes are known to contain high levels