Zobrazeno 1 - 10
of 515
pro vyhledávání: '"J P Cartron"'
Autor:
Marie-France Mamzer, T. Sannié, J.-P. Cartron, Antoine Haddad, M.-C. Bouësseau, B. Clavier, S. Noël, Christian Hervé, Jean-Baptiste Thibert, M. Monsellier, Jean-Daniel Tissot, J. Ceccaldi, Bruno Danic, R. Pottier, J. P. Vernant, Olivier Garraud
Publikováno v:
Transfusion Clinique et Biologique. 24:76-82
The not-for-profit issue has been debated in November 2016 in Paris; this issue is one of the four canonical pillars of ethical blood donation. It is intimately bound to benevolence though it is distinct, as not-for-profit calls for institutions whil
Autor:
Jacques Chiaroni, J. P. Vernant, Bernard Pelletier, M. Monsellier, B Cuneo, M-A Hermitte, S Mackowiak, Olivier Garraud, A Saillol, J.-P. Cartron, J-D Tissot, S Moreau, Bruno Danic, Cécile Hervé, R. Pottier, M Guimelchain-Bonnet, K Papa, B. Clavier, R Praile
Publikováno v:
Transfusion Clinique et Biologique. 23:168-174
Voluntariness stands for one of the four pillars of ethics in blood donation; it is, however, more related to tradition than to legislation. Because it seems necessary to apply "marketing" techniques to blood collection in order to meet the needs in
Autor:
J, Ceccaldi, J-B, Thibert, A, Haddad, M-C, Bouësseau, R, Pottier, B, Danic, S, Noël, M, Monsellier, J-D, Tissot, T, Sannié, B, Clavier, M-F, Mamzer, J-P, Cartron, J-P, Vernant, C, Hervé, O, Garraud
Publikováno v:
Transfusion clinique et biologique : journal de la Societe francaise de transfusion sanguine. 24(2)
The not-for-profit issue has been debated in November 2016 in Paris; this issue is one of the four canonical pillars of ethical blood donation. It is intimately bound to benevolence though it is distinct, as not-for-profit calls for institutions whil
Autor:
J.-P. Cartron
Publikováno v:
ISBT Science Series. 5:27-45
Numerous studies in biochemistry, genetics and molecular biology conducted over two decades have shown that blood group gene products expressed on red cells and various tissues exhibit a variety of potential functions, which can be schematically clas
Autor:
W. El Nemer, M.-P. Wautier, C. Le Van Kim, Cécile Rahuel, J.-P. Cartron, Anne Filipe, J.-L. Wautier, Yves Colin
Publikováno v:
Transfusion Clinique et Biologique. 15:402-405
Recent studies shed new lights on the biological function of blood group antigens, such as the adhesion properties of the Lutheran (Lu) blood group antigens carried by the Lu/BCAM glycoproteins. The Lu/BCAM adhesion glycoproteins were first identifie
Autor:
J.-P. Cartron, Jacques Elion
Publikováno v:
Transfusion Clinique et Biologique. 15:39-50
In sickle cell disease, the complex scenario of vaso-occlusive crisis (VOC) typical of this disease is clearly multifactorial and not fully understood. Cell-cell and cell-cell matrix interactions mediated by adhesive molecules present on blood cells
Autor:
M.-P. Wautier, J.-P. Cartron, Yves Colin, W. El Nemer, C. Le Van Kim, Pierre Gane, Emilie Gauthier, Cécile Rahuel, Frédéric Galactéros, J.-L. Wautier
Publikováno v:
Transfusion Clinique et Biologique. 15:29-33
Lutheran (Lu) blood group and Basal Cell Adhesion Molecule (BCAM) antigens are both carried by two glycoprotein (gp) isoforms of the immunoglobulin superfamily representing receptors for laminin alpha5 chain. They are expressed in red blood cells, in
Autor:
C. Rouillac-Le Sciellour, Yves Colin, C. Le Van Kim, Y. Brossard, J.-P. Cartron, Y. Guidicelli, Valérie Serazin, O. Oudin, M. Menu
Publikováno v:
Transfusion Clinique et Biologique. 14:572-577
Fetal RHD genotyping from maternal plasma was performed by real-time PCR amplification of exons 7 and 10 of the RHD gene and the amplified products were detected either with SYBR Green I dye according to our previously published method [Mol Diagn 8 (
Autor:
Philippe Rouger, Stéphanie Martin-Blanc, P.Y. Le Pennec, Christophe Tournamille, J.-P. Cartron, Hélène Ansart-Pirenne
Publikováno v:
Vox Sanguinis. 92:142-147
Background and Objectives The Duffy (FY) blood group system is controlled by four major alleles: FY*A and FY*B, the Caucasian common alleles, encoding Fya and Fyb antigens; FY*X allele responsible for a poorly expressed Fyb antigen, and FY*Fy a silen