Zobrazeno 1 - 10
of 18
pro vyhledávání: '"J J, Stragand"'
Publikováno v:
The Journal of nutrition. 126(8)
To assess the effects of guar gum on colonic microbial fermentation and cancer development, azoxymethane-treated rats were fed a partially hydrolyzed guar or control diet. Anaerobic fecal incubations were conducted at 8-wk intervals, either without a
Autor:
J. J. Stragand, Ronald F. Hagemann
Publikováno v:
Cell Proliferation. 10:3-14
Following a period of fasting, feeding a normal diet results in a burst of DNA synthesis in the crypts of the colonic epithelium. This is due largely to a prompt entry of cells, blocked in G1, into S. Peak levels of S cellularity exceed 4 times the f
Publikováno v:
British Journal of Cancer
The cytotoxic and stathomokinetic effects of vincristine (VCR) on murine fibrosarcoma (FSa) cells, grown either in vitro as primary cultures or in vivo as micro- or macroscopic pulmonary nodules, were determined and compared. FSa cells were separated
Publikováno v:
Cancer. 43:1707-1719
The cellular kinetics of human mammary tumors were studied by in vitro methods. These techniques include single 3HTdR labeling to measure the 3HTdR LI, double labeling with 3HTdR and 14CTdR to measure DNA synthesis time, and an estimation of the grow
Publikováno v:
Antibiotics and chemotherapy. 23
Autor:
J J Stragand, R F Hagemann
Publikováno v:
The American journal of clinical nutrition. 30(6)
Publikováno v:
Cancer research. 38(12)
An iron requirement for the synchronous progression of colonic cells following fasting and refeeding
Autor:
J. J. Stragand, Ronald F. Hagemann
Publikováno v:
Cell and tissue kinetics. 11(5)
Evidence is presented to show dietary iron to be a major co-factor in the colonic hyperplasia observed following fasting and refeeding. The iron component serves to remove a fasting induced colonic G1 cycle block and produce the resultant synchronous
Autor:
J. J. Stragand, R. F. Hagemann
Publikováno v:
Experientia. 34(2)
Following fasting and refeeding, the colonic epithelium of the rat exhibits a marked hyperplasia. This response is of a similar magnitude but of a longer duration to that observed in mice. This response is not affected by reducing serum calcium level