Zobrazeno 1 - 10
of 61
pro vyhledávání: '"J E Foley"'
Publikováno v:
Irish Journal of Psychological Medicine. 36:145-151
AimTo investigate whether a small regional memory clinic would benefit from engaging with a structured external audit process such as the Royal College of Psychiatrists’ Memory Service National Accreditation Program (MSNAP).BackgroundThe Psychiatry
Autor:
Eva Ekblad, Bilal Omar, Bo Ahrén, Ulrikke Voss, J E Foley, Maria Sörhede Winzell, Jenny Vikman
Publikováno v:
Diabetologia. 56:1752-1760
Studies have shown that dipeptidyl peptidase-4 (DPP4) inhibitors stimulate insulin secretion and increase beta cell mass in rodents. However, in these models hyperglycaemia has been induced early on in life and the treatment periods have been short.
Publikováno v:
Diabetic Medicine. 25:435-441
Aims To compare the efficacy and tolerability of the dipeptidyl peptidase-4 inhibitor, vildagliptin, with the alpha glucosidase inhibitor, acarbose, in drug-naive patients with Type 2 diabetes. Methods This multi-centre, randomized, double-blind, par
Publikováno v:
International Journal of Clinical Practice. 62:8-14
Vildagliptin is a potent, selective and reversible inhibitor of dipeptidyl peptidase-4 (DPP-4), the enzyme responsible for rapid inactivation of the incretin hormones glucagon-like peptide-1 (GLP-1) and gastric inhibitory polypeptide (GIP). GLP-1 and
Publikováno v:
Hormone and metabolic research 39 (2007): 826–829.
info:cnr-pdr/source/autori:Ahren B, Pacini G, Tura A, Foley JE and Schweizer A,/titolo:Improved meal-related insulin processing contributes to the enhancement of B-cell function by the DPP-4 inhibitor vildagliptin in patients with type 2 diabetes/doi:/rivista:Hormone and metabolic research/anno:2007/pagina_da:826/pagina_a:829/intervallo_pagine:826–829/volume:39
info:cnr-pdr/source/autori:Ahren B, Pacini G, Tura A, Foley JE and Schweizer A,/titolo:Improved meal-related insulin processing contributes to the enhancement of B-cell function by the DPP-4 inhibitor vildagliptin in patients with type 2 diabetes/doi:/rivista:Hormone and metabolic research/anno:2007/pagina_da:826/pagina_a:829/intervallo_pagine:826–829/volume:39
The aim of this study was to evaluate the contribution of insulin processing to the improved meal-related B-cell function previously shown with the DPP-4 inhibitor vildagliptin. Fifty-five patients with type 2 diabetes (56.5+/-1.5 years; BMI=29.6+/-0
Publikováno v:
Diabetologia. 48:1700-1713
Although there is abundant evidence that hyperglucagonaemia plays a key role in the development of hyperglycaemia in type 2 diabetes, efforts to understand and correct this abnormality have been overshadowed by the emphasis on insulin secretion and a
Publikováno v:
Diabetes, obesitymetabolism. 16(9)
To determine the effects of dipeptidyl peptidase-4 (DPP-4) inhibition on glucagon dynamics in patients with insulin-treated type 2 diabetes (T2D).The study was a single-centre, double-blind, randomized, placebo controlled crossover study in patients
Several new oral antidiabetic agents, known as 'gliptins' or 'enzyme dipeptidyl peptidase-IV (DPP-4) inhibitors', have been developed for the treatment of type 2 diabetes and a key clinical use of the gliptins is in combination with metformin. There
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c82f5201132c3013f62489812f6757c8
Autor:
B. Zinman, D. R. Matthews, Bo Ahrén, S. Dejager, Vivian Fonseca, J. E. Foley, A. Couturier, Ele Ferrannini
Publikováno v:
Diabetes, obesitymetabolism. 12(9)
AIM: To show that vildagliptin added to metformin is non-inferior to glimepiride in reducing HbA1c levels from baseline over 2 years. METHODS: A randomized, double-blind, active-comparator study of patients with type 2 diabetes mellitus inadequately
Autor:
J E, Sykes, S L, Marks, S, Mapes, R M, Schultz, R E, Pollard, D, Tokarz, P P, Pesavento, L A, Lindsay, J E, Foley
Publikováno v:
Journal of veterinary internal medicine. 24(3)
Salmon poisoning disease (SPD) is a trematode-borne disease of dogs caused by Neorickettsia helminthoeca.To determine risk factors and spatial epidemiology of SPD in dogs from northern California; to describe the clinicopathologic, microbiologic, and