Zobrazeno 1 - 10
of 76
pro vyhledávání: '"Jürgen Kartenbeck"'
Autor:
Eric Ghigo, Jürgen Kartenbeck, Pham Lien, Lucas Pelkmans, Christian Capo, Jean-Louis Mege, Didier Raoult
Publikováno v:
PLoS Pathogens, Vol 4, Iss 6, p e1000087 (2008)
Mimivirus, or Acanthamoeba polyphaga mimivirus (APMV), a giant double-stranded DNA virus that grows in amoeba, was identified for the first time in 2003. Entry by phagocytosis within amoeba has been suggested but not demonstrated. We demonstrate here
Externí odkaz:
https://doaj.org/article/a1ece606bfe14dfc9ab0890eef715f97
Autor:
Roberta Mancini, Jürgen Kartenbeck, Ari Helenius, Fabian Herzog, Sabrina Engel, Thomas Heger, Arnold Hayer
After binding to its cell surface receptor ganglioside GM1, simian virus 40 (SV40) is endocytosed by lipid raft-mediated endocytosis and slowly transported to the endoplasmic reticulum, where partial uncoating occurs. We analyzed the intracellular pa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9ca656d1a8baec4fa652acf5e6eb4a15
https://europepmc.org/articles/PMC5625483/
https://europepmc.org/articles/PMC5625483/
Publikováno v:
European Journal of Cell Biology. 84:417-430
Various authors have reported reduced synthesis of epithelial junctional proteins during dedifferentiation, tumorigenesis and metastasis in a great variety of tumors. Consequently, it is generally accepted that loss of adhesive molecules and adhesion
Autor:
Anna Mezzacasa, Eva-Maria Damm, Teymuras V. Kurzchalia, Lucas Pelkmans, Jürgen Kartenbeck, Ari Helenius
Publikováno v:
The Journal of Cell Biology
Simian Virus 40 (SV40) has been shown to enter host cells by caveolar endocytosis followed by transport via caveosomes to the endoplasmic reticulum (ER). Using a caveolin-1 (cav-1)–deficient cell line (human hepatoma 7) and embryonic fibroblasts fr
Publikováno v:
International Journal of Cancer. 114:779-790
E-cadherin-mediated and desmosomal cell-cell adhesion have been implicated in the suppression of invasive and metastatic behavior of squamous cell carcinomas. Whether the adhaerens junction represented by E-cadherin and the desmosomes interplay or ha
Autor:
Birgit Ottenwälder, Nikolaus Gassler, Herwart F. Otto, Jürgen Kopitz, Annemarie Poustka, Frank Autschbach, Arman Tehrani, Jan Mollenhauer, Stefan Lyer, Martina Schnölzer, Jürgen Kartenbeck
Publikováno v:
The Journal of Pathology. 202:188-196
Several disorders of the small intestine are associated with disturbances in villus architecture. Thus, an understanding of the molecular mechanisms associated with the differentiation of villi represents an important step in the improvement of the u
Autor:
Werner A. Eckert, Jürgen Kartenbeck
Proteine stellen die vielfältigsten Moleküle lebender Organismen dar und spielen bei praktisch allen biologischen Strukturen und Abläufen eine zentrale Rolle. Daher sind Methoden zur Isolierung und Charakterisierung von Proteinen die Grundlage jed
Autor:
Frank Autschbach, Herwart F. Otto, Armin Schneider, Nikolaus Gassler, Nicholas Obermüller, Martina Schnölzer, Jürgen Kartenbeck, Jürgen Kopitz
Publikováno v:
Human Pathology. 34:1048-1052
Fatty acids are implicated in tumorigenesis, but data are limited concerning endogenous fatty acid metabolism of tumor cells in adenomas and adenocarcinomas of the small intestine. The recently cloned human acyl-CoA-synthetase 5 (ACS5) is predominant
Autor:
Ivo C. Lorenz, Anna Mezzacasa, Ari Helenius, Jürgen Kartenbeck, Franz X. Heinz, Steven L. Allison
Publikováno v:
Journal of Virology. 77:4370-4382
It is believed that flavivirus assembly occurs by intracellular budding of the nucleocapsid into the lumen of the endoplasmic reticulum (ER). Recombinant expression of tick-borne encephalitis (TBE) virus envelope proteins prM and E in mammalian cells
Autor:
Angel Alonso, Jürgen Kartenbeck, Margarita M. Marques, Hao Cheng, Kirsten Kabsch, Xiahong Mao
Publikováno v:
Journal of Cellular Physiology. 192:234-243
Activation of the epidermal growth factor receptor (EGFR) has been shown to occur by ligand-dependent and ligand-independent mechanisms. Different molecular mechanisms have been found to be responsible for ligand-independent receptor transactivation.