Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Júlia Kurkó"'
Autor:
Júlia Kurkó, András Vida, Tímea Ocskó, Beata Tryniszewska, Tibor A Rauch, Tibor T Glant, Zoltán Szekanecz, Katalin Mikecz
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e111815 (2014)
Myeloid-derived suppressor cells (MDSCs) are innate immune cells capable of suppressing T-cell responses. We previously reported the presence of MDSCs with a granulocytic phenotype in the synovial fluid (SF) of mice with proteoglycan (PG)-induced art
Externí odkaz:
https://doaj.org/article/220eedaecf3a4d2cb43cb713ab9d9e19
Autor:
Steve Cress, Roy E. Carambula, Júlia Kurkó, Ken S. Rosenthal, Harold L. Steiner, Daniel H. Zimmerman, Katalin Mikecz, Adrienn Markovics, Tibor T. Glant
Publikováno v:
Vaccine. 35(32)
Rheumatoid arthritis (RA) is an autoimmune joint disease maintained by aberrant immune responses involving CD4+ T helper (Th)1 and Th17 cells. In this study, we tested the therapeutic efficacy of Ligand Epitope Antigen Presentation System (LEAPS™)
Autor:
Tibor A. Rauch, Timea Besenyei, Robert S. Katz, János Gál, Beata Tryniszewska, Katalin Mikecz, András Kádár, Györgyike Soós, Júlia Kurkó, Zoltán Szekanecz, Tibor T. Glant, Joel A. Block, Gyula Hoffmann
Publikováno v:
Arthritis & Rheumatism. 65:1725-1735
Objective To identify epigenetic factors that are implicated in the pathogenesis of rheumatoid arthritis (RA), and to explore the therapeutic potential of the targeted inhibition of these factors. Methods Polymerase chain reaction (PCR) arrays were u
Autor:
Beata Tryniszewska, Tibor A. Rauch, András Vida, Tímea Ocskó, Katalin Mikecz, Tibor T. Glant, Júlia Kurkó, Zoltán Szekanecz
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e111815 (2014)
PLoS ONE
PLoS ONE
Background Myeloid-derived suppressor cells (MDSCs) are innate immune cells capable of suppressing T-cell responses. We previously reported the presence of MDSCs with a granulocytic phenotype in the synovial fluid (SF) of mice with proteoglycan (PG)-
Autor:
Timea Besenyei, Júlia Kurkó, Judit Laki, Enikö Simkovics, Andrea Váncsa, Laszlo Nagy, Edit Végh, Tibor T. Glant, Szilvia Szamosi, Szilárd Póliska, Katalin Mikecz, Bertalan Meskó, Zoltán Szekanecz
Pharmacogenetics and pharmacogenomics deal with possible associations of a single genetic polymorphism or those of multiple gene profiles with responses to drugs. In rheumatology, genes and gene signatures may be associated with altered efficacy and/
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b77487995a14637f8f59844186b2247f
The "Bermuda triangle" of genetics, environment and autoimmunity is involved in the pathogenesis of rheumatoid arthritis (RA). Various aspects of genetic contribution to the etiology, pathogenesis and outcome of RA are discussed in this review. The h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b8812373570a90de28919e4e34d3a20e
Autor:
Tibor T. Glant, Beata Tryniszewska, András Kádár, Timea Besenyei, Katalin Mikecz, Júlia Kurkó, Tibor A. Rauch, Zoltán Szekanecz
Publikováno v:
Clinical and Developmental Immunology, Vol 2012 (2012)
Clinical and Developmental Immunology
Clinical and Developmental Immunology
Rheumatoid arthritis (RA) is a polygenic autoimmune disease primarily affecting the synovial joints. Numerous animal models show similarities to RA in humans; some of them not only mimic the clinical phenotypes but also demonstrate the involvement of
Autor:
András Vida, Júlia Kurkó, Tibor T. Glant, Robert S. Katz, Anjali Nair, Zoltán Szekanecz, Katalin Mikecz, Carla R. Scanzello
Publikováno v:
BMC Musculoskeletal Disorders
Background Myeloid-derived suppressor cells (MDSCs) are a heterogeneous population of innate immune cells with a granulocyte-like or monocyte-like phenotype and a unique ability to suppress T-cell responses. MDSCs have been shown to accumulate in can
Autor:
Timea Besenyei, Andras Kadar, Beata Tryniszewska, Julia Kurko, Tibor A. Rauch, Tibor T. Glant, Katalin Mikecz, Zoltan Szekanecz
Publikováno v:
Clinical and Developmental Immunology, Vol 2012 (2012)
Rheumatoid arthritis (RA) is a polygenic autoimmune disease primarily affecting the synovial joints. Numerous animal models show similarities to RA in humans; some of them not only mimic the clinical phenotypes but also demonstrate the involvement of
Externí odkaz:
https://doaj.org/article/ea70ac6441d9482888cdaff6d7a961fd