Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jørn B. Sande"'
Autor:
Ivar Sjaastad, Jørn B. Sande, Muthu Periasamy, William E. Louch, Halvor K. Mørk, Ole M. Sejersted
Publikováno v:
Journal of Molecular and Cellular Cardiology. 43:177-186
End-stage heart failure is believed to involve depressed cardiomyocyte contractility and Ca2+ transients. However, the time course of these alterations is poorly understood. We examined alterations in myocyte excitation-contraction coupling in a mous
Autor:
Ingvild B Hoen, Ivar Sjaastad, Per Kristian Lunde, Janny Bøkenes, Geir Christensen, Theis Tønnessen, Jørn B. Sande, Even Holt
Publikováno v:
Cardiovascular Research. 53:382-391
Objective: Heart failure is associated with alterations in contractile parameters and accompanied by abnormalities in intracellular calcium homeostasis. Sarcoplasmic reticulum Ca2+ ATPase (SERCA2) and phospholamban (PLB) are important in intracellula
Autor:
Theis Tønnessen, Tævje A. Strømme, Jørn B. Sande, Torstein Lyberg, Per Kristian Lunde, Srdjan Djurovic, Per Reidar Woldbaek, Geir Christensen
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 289(2)
Although increased levels of circulating interleukin (IL)-18 have been demonstrated in patients with cardiovascular diseases, the functional consequences of chronically increased circulating IL-18 with respect to myocardial function have not been def
Publikováno v:
Advances in Electrocardiology 2004.
Autor:
Per Grøttum, William E. Louch, Jørn B. Sande, Halvor K. Mørk, Ole M. Sejersted, Glenn T. Lines
Publikováno v:
Biophysical Journal. (3):779-792
Trigger Ca(2+) is considered to be the Ca(2+) current through the L-type Ca(2+) channel (LTCC) that causes release of Ca(2+) from the sarcoplasmic reticulum. However, cell contraction also occurs in the absence of the LTCC current (I(Ca)). In this ar