Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Jéssica Assis Pereira"'
Autor:
Vanessa Fernandes Rodrigues, Jefferson Elias-Oliveira, Ítalo Sousa Pereira, Jéssica Assis Pereira, Sara Cândida Barbosa, Melissa Santana Gonsalez Machado, Daniela Carlos
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Akkermansia muciniphila is a Gram-negative anaerobic mucus-layer-degrading bacterium that colonizes the intestinal mucosa of humans and rodents. Metagenomic data have shown an inverse correlation between the abundance of A. muciniphila and diseases s
Externí odkaz:
https://doaj.org/article/a0a6c97a615e411bb0e1777a64335545
Autor:
Aleksander Brandão Santana, Bianca Silva Souto, Natália Cristina de Melo Santos, Jéssica Assis Pereira, Carlos Alberto Tagliati, Rômulo Dias Novaes, Patrícia Paiva Corsetti, Leonardo Augusto de Almeida
Publikováno v:
Life sciences. 307
This manuscript aims to explain the relationship between mucositis caused by 5-FU over gut bacterial species and susceptibility to opportunistic infection caused by P. aeruginosa.BALB/c mice were intraperitoneally treated with PBS or 5-FU. Bodyweight
Autor:
Caio Pupin Rosa, Leonardo A. de Almeida, Carlos Alberto Tagliati, Gustavo Andrade Brancaglion, Natália Cristina de Melo Santos, Jéssica Assis Pereira, Rômulo Dias Novaes, Patricia P. Corsetti, Evandro Neves Silva
Publikováno v:
Journal of Leukocyte Biology. 107:95-104
Pseudomonas aeruginosa is one of the most common opportunistic pathogens causing respiratory infections in hospitals. Vancomycin, the antimicrobial agent usually used to treat bacterial nosocomial infections, is associated with gut dysbiosis. As a lu
Autor:
Bruna Pinheiro Pereira, Rômulo Dias Novaes, Leonardo A. de Almeida, Valéria Aparecida Belo, Carla S. Ceron, Débora de Oliveira Lopes, Patricia P. Corsetti, Jéssica Assis Pereira, Sara Franchin D Souza, Fernanda de Lima Tana
Publikováno v:
Cell and tissue research. 383(3)
Pseudomonas aeruginosa is considered an opportunistic pathogen of great clinical importance. The clearance of this bacterium occurs through recognition of the pathogen by innate immune system receptors, leading to a lung inflammatory response. Howeve