Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Ivo Florin Scheiber"'
Autor:
Matyas Krijt, Emmanuel Lesuisse, Thibaut Léger, Jean-Michel Camadro, Ronald Malych, Jan Mach, Eva Kotabová, Daniel Vyoral, Ivo Florin Scheiber, Jana Pilátová, Robert Sutak, Ondřej Prášil
Publikováno v:
Metallomics
Metallomics, Royal Society of Chemistry, 2019, 11 (10), pp.1657-1666. ⟨10.1039/c9mt00078j⟩
Metallomics, Royal Society of Chemistry, 2019, 11 (10), pp.1657-1666. ⟨10.1039/c9mt00078j⟩
International audience; Iron and copper are essential elements for practically all living organisms. Their metabolism is frequently interconnected, and while copper is relatively abundant in the ocean, iron is often a limiting factor for the growth o
Publikováno v:
Nutrients
Nutrients, Vol 11, Iss 7, p 1473 (2019)
Volume 11
Issue 7
Nutrients, Vol 11, Iss 7, p 1473 (2019)
Volume 11
Issue 7
The alveolar epithelia of the lungs require manganese (Mn) as an essential nutrient, but also provide an entry route for airborne Mn that can cause neurotoxicity. Transporters involved in Mn uptake by alveolar epithelial cells are unknown. Recently,
Publikováno v:
J Biol Chem
ZIP14 (encoded by the solute carrier 39 family member 14 (SLC39A14) gene) is a manganese transporter that is abundantly expressed in the liver and small intestine. Loss-of-function mutations in SLC39A14 cause severe hypermanganesemia. Because the liv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2807d960e9e6efc44dee434727684c88
https://europepmc.org/articles/PMC6556583/
https://europepmc.org/articles/PMC6556583/
Publikováno v:
Advances in neurobiology. 18
Copper is an essential trace metal that is required for several important biological processes, however, an excess of copper can be toxic to cells. Therefore, systemic and cellular copper homeostasis is tightly regulated, but dysregulation of copper
Publikováno v:
Handbook of clinical neurology. 142
Wilson disease is an autosomal-recessive disorder originating from a genetic defect in the copper-transporting ATPase ATP7B that is required for biliary copper secretion and loading of ceruloplasmin with copper. Impaired ATP7B function in Wilson dise