Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Ivana Fantozzi"'
Autor:
Eun A. Ko, Ivana Fantozzi, Amy L. Firth, Oleksandr Platoshyn, Jason X.-J. Yuan, Carmelle V. Remillard
Publikováno v:
Pulmonary Circulation
The activity of voltage-gated ion channels is critical for the maintenance of cellular membrane potential and generation of action potentials. In turn, membrane potential regulates cellular ion homeostasis, triggering the opening and closing of ion c
Autor:
Ada H. Wong, Jason X.-J. Yuan, Manohar R. Furtado, Ivana Fantozzi, Shen Zhang, Oleksandr Platoshyn, Olga Petrauskene, Carmelle V. Remillard
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 291:L993-L1004
Activity of voltage-gated K+(KV) channels in pulmonary artery smooth muscle cells (PASMC) plays an important role in control of apoptosis and proliferation in addition to regulating membrane potential and pulmonary vascular tone. Bone morphogenetic p
Publikováno v:
Pflügers Archiv - European Journal of Physiology. 451:380-387
Electrical excitability, which plays an important role in excitation-contraction coupling in the pulmonary vasculature, is regulated by transmembrane ion flux in pulmonary artery smooth muscle cells (PASMC). This study aimed to characterize the elect
Autor:
Oleksandr Platoshyn, Shen Zhang, Jason X.-J. Yuan, Patricia A. Thistlethwaite, Ivana Fantozzi, Wei Huang, Jifeng Zhang, Carmelle V. Remillard
Publikováno v:
Experimental Lung Research. 31:783-806
Bone morphogenetic proteins (BMPs) inhibit proliferation and induce apoptosis in pulmonary artery smooth muscle cells (PASMCs) from normal subjects. Dysfunction of BMP signaling due to mutations in and/or down-regulation of BMP receptors has been imp
Autor:
Shen Zhang, Ivana Fantozzi, Carmelle V. Remillard, Jason X.-J. Yuan, Oleksandr Platoshyn, Randy T. Cowling
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 285:L1233-L1245
Activating protein (AP)-1 transcription factors modulate expression of genes involved in cell proliferation and migration. Chronic hypoxia increases pulmonary artery smooth muscle cell proliferation by upregulating AP-1-responsive genes encoding for
Autor:
Michele De Luca, Graziella Pellegrini, Diego Ponzin, Ivana Fantozzi, Elena Dellambra, Frank McKeon, Enrica Martinelli, Osvaldo Golisano, Sergio Bondanza
Publikováno v:
Proceedings of the National Academy of Sciences. 98:3156-3161
The proliferative compartment of stratified squamous epithelia consists of stem and transient amplifying (TA) keratinocytes. Some polypeptides are more abundant in putative epidermal stem cells than in TA cells, but no polypeptide confined to the ste
Autor:
Sébastien Cagnol, Fabio Stanchi, Gérard Milano, Ellen Van Obberghen-Schilling, Marie-Christine Brunstein, Dominique Grall, Alexandre Bozec, Anne Sudaka, Ivana Fantozzi, Jean-Louis Fischel
Publikováno v:
Langenbeck's Archives of Surgery
Langenbeck's Archives of Surgery, Springer Verlag, 2008, pp.1-11. ⟨10.1080/02841860802089801⟩
Langenbeck's Archives of Surgery, Springer Verlag, 2008, pp.1-11. ⟨10.1080/02841860802089801⟩
Background. The gene encoding cortactin, CTTN (locus 11q13), an actin-binding substrate of Src kinases, is frequently amplified in breast and head and neck squamous cell carcinomas (HNSCC) and cortactin overexpression is thought to contribute in a si
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::669ea7cd5f14d9285934157de391e91a
https://hal.archives-ouvertes.fr/hal-00319334
https://hal.archives-ouvertes.fr/hal-00319334
Publikováno v:
FEMS yeast research. 7(5)
KlADH3 is a Kluyveromyces lactis alcohol dehydrogenase gene induced in the presence of all respiratory carbon sources except ethanol, which specifically represses this gene. Deletion analysis of the KlADH3 promoter revealed the presence of both posit
Autor:
Ying Yu, Judd W. Landsberg, Jason X.-J. Yuan, Patricia A. Thistlethwaite, Oleksandr Platoshyn, Ivana Fantozzi, Carmelle V. Remillard, Naomi Kunichika, Lewis J. Rubin, Donna D. Tigno
Pulmonary vascular medial hypertrophy caused by excessive pulmonary artery smooth muscle cell (PASMC) proliferation is a major cause for the elevated pulmonary vascular resistance in patients with idiopathic pulmonary arterial hypertension (IPAH). In
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc355aa50cbe4058068bac063d871474
https://europepmc.org/articles/PMC518765/
https://europepmc.org/articles/PMC518765/
Publikováno v:
American journal of physiology. Cell physiology. 287(5)
Extracellular ATP and intracellular cyclic AMP response element-binding protein (CREB, a transcription factor) promote cell proliferation in many cell types. The canonical transient receptor potential (TRPC) channels, which putatively participate in