Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ivan Sammir Aranda-Uribe"'
Autor:
Silvia Jiménez-Morales, Ivan Sammir Aranda-Uribe, Carlos Jhovani Pérez-Amado, Julian Ramírez-Bello, Alfredo Hidalgo-Miranda
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Acute lymphoblastic leukemia (ALL) is a malignancy with high heterogeneity in its biological features and treatments. Although the overall survival (OS) of patients with ALL has recently improved considerably, owing to the application of conventional
Externí odkaz:
https://doaj.org/article/a5fa3f0c953e4009bd4659fe726e9991
Publikováno v:
Autoimmunity, Vol 50, Iss 6, Pp 336-345 (2017)
The breakdown of immunological tolerance due to the activation of autoreactive B and T cells triggers physiopathological processes. An example of such conditions is the production of IgG autoantibodies specific for the Fc portion of IgG (anti-Fcγ Ig
Externí odkaz:
https://doaj.org/article/8cd09686d73b44f89cec64d2de1f56e5
Autor:
Rosa Elda Barbosa-Cobos, Julian Ramírez-Bello, Juan Carlos López-Vázquez, Ivan Sammir Aranda-Uribe
Publikováno v:
Molecular Biology Reports. 48:3561-3565
Toll-like receptor (TLR)-mediated signaling pathways induce a proinflammatory microenvironment to eradicate pathogens. However, in rheumatoid arthritis (RA) and systemic lupus erythematosus (SLE), TLRs can promote chronic inflammation. It has been sh
Autor:
Julian Ramírez-Bello, Ivan Sammir Aranda-Uribe, José Moreno, Georgina Victoria-Acosta, Rosa Elda Barbosa-Cobos
Rheumatoid arthritis (RA) is a chronic inflammatory disease of autoimmune origin associated with many genetic traits, including genes related to the control of inflammation. The A20 protein, encoded by the TNFAIP3 gene, is a negative regulator of NF-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::36921be73269aef1dbbee181702fd8f3
https://doi.org/10.21203/rs.3.rs-1039113/v1
https://doi.org/10.21203/rs.3.rs-1039113/v1
Autor:
Ivan Sammir Aranda-Uribe, Julian Ramírez-Bello, Georgina Victoria-Acosta, Felipe Muñoz-González, Rosa Elda Barbosa-Cobos, José Moreno
Publikováno v:
Molecular biology reports. 49(11)
Rheumatoid arthritis (RA) is a chronic inflammatory disease of autoimmune origin with many associated genetic traits, including genes related to the control of inflammation. The A20 protein, encoded by the TNFAIP3 gene, is a negative regulator of NF-
Autor:
José Manuel Fragoso, Julian Ramírez-Bello, Mario Adán Moreno-Eutimio, Guillermo Aquino-Jarquin, Carmen Estefanía Martínez-Alemán, Rosa Elda Barbosa-Cobos, Carlos Cabello-Gutierrez, Miguel A. Saavedra, Ivan Sammir Aranda-Uribe
Publikováno v:
Clinical rheumatology. 40(3)
The aim of this study was to examine the association of three TNFSF4 single nucleotide variants (SNVs) with systemic lupus erythematosus (SLE) susceptibility in Mexican patients.Genotypes of the TNFSF4 rs1234315T/C, rs2205960G/T, and rs704840T/G SNVs
Publikováno v:
Autoimmunity. 50(6)
The breakdown of immunological tolerance due to the activation of autoreactive B and T cells triggers physiopathological processes. An example of such conditions is the production of IgG autoantibodies specific for the Fc portion of IgG (anti-Fcγ Ig
Autor:
Toriello Concepcion, Rafael Laniado-Laborín, Arturo Aroch-Calderón, Amelia Pérez-Mejía, María del Rocío González-Martínez, María Eugenia Manjarrez-Zavala, Ivan Sammir Aranda-Uribe, Alfonso Hernández-Navarez, Gabriel Palma, María Antonieta Flores-Sánchez, Bertha Muñoz-Hernández, Sandra Miranda-Mauricio, Laura Rosio Castañón-Olivares
Publikováno v:
Mycopathologia. 169(6)
Coccidioidin, an extract from the saprophytic mycelial form of Coccidioides spp., has been a very useful antigen preparation both for skin and serological tests for coccidioidomycosis. Unfortunately, coccidioidin is not currently available for skin t